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New mutations in the SLC39A4 gene found in twins help understand the genetic cause of acrodermatitis enteropathica.

African spiny mice can regenerate skin, hair, and cartilage, but not muscle, and their unique abilities could be useful for regenerative medicine.

Mice without certain skin proteins had abnormal skin and hair development.

Overexpression of the glucocorticoid receptor in mice causes developmental defects similar to ectodermal dysplasia.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

The Naked (N) trait in mice is linked to lower glycine and tyrosine in hair proteins.

Normal cells outcompete and remove mutant cells in the pancreas with the help of the EphA2 receptor.

Retinoic acid causes gland formation instead of hair in mouse skin by altering epidermal and dermal interactions.

CRISPR/Cas9 and prime editing can potentially fix skin disorder genes safely and effectively.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

Rabbits lacking the Hoxc13 gene show similar hair and skin issues to humans with ECTD-9, making them good for research on this condition.

Mus pahari mice have fragile skin due to abnormal collagen and elastin.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

Activating Kras in mouse skin causes excess skin and hair loss.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

A mouse model was created to study hair loss similar to humans.

CD8+ T cells expand significantly in alopecia areata, suggesting new treatment targets.

Hair follicles in the back of the rosette fancy mouse have reversed orientations due to a gene mutation.

Msx2 deficiency in mice leads to bone growth and organ development problems.

A gene deletion causes the "hairless" trait in Iffa Credo rats.

Mice with a changed Hr gene lose and regrow hair due to changes in the gene's activity.

Injecting certain cells into mice caused hair loss, which was preventable with a specific inhibitor.

Gene knockout mice developed scars similar to human hypertrophic scars, useful for studying scar progression.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

Keratin2-6g is crucial for hair follicle development, with mutations causing cell degeneration and vacuolation.

Researchers fixed gene mutations causing a skin disease in stem cells, which then improved skin grafts in mice.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

The mutation helps mice handle heat better without affecting hair growth.

Mutant Cx43 causes slower wound healing and hair growth issues in ODDD.