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A specific group of immune and skin cells may cause chronic inflammation in atopic dermatitis.

Psoriasis may be chronic because it lacks certain immune system controls that prevent overreaction.

Melanocytes may trigger the immune response in alopecia areata, affecting hair regrowth.

γδ T cells are crucial for early wound healing after a skin virus infection.

Highly active but fewer CD14+CD16- monocytes are found in Alopecia Areata patients, regardless of severity.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

Higher levels of certain immune cells in hair follicles may contribute to alopecia areata.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

Targeting CD169+ skin macrophages may help treat psoriasis.

Natural killer cells play a key role in autoimmune skin diseases like pemphigus vulgaris.

HTLV-1-associated lichenoid dermatitis (HALD) is linked to an immune response against HTLV-1-infected cells.

MAIT cells may help fight COVID-19 but also contribute to severe inflammation.

Type-2 immunity may influence skin diseases and could be targeted for treatment.

Cutaneous lupus patients have higher levels of certain immune cells in their blood and skin.

ICL is a condition with low CD4+ T cells like AIDS but not caused by HIV, and normal CD4+ T cell counts may vary between men and women.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

IL-2 treatment causes skin eruptions and other reversible side effects, and may play a role in psoriasis.

Regulatory T-cells are important for healing and regenerating tissues in various organs by controlling immune responses and aiding stem cells.

A new type of nerve cell involved in itch perception was discovered.

Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

B cells can both help and hinder the body's defense against melanoma.

Farudodstat can prevent hair follicle immune damage linked to alopecia areata.

Human thymus has stem cells that can self-renew and maintain their identity.

Sparse hairless patches can develop and stabilize in alopecia areata under certain conditions.

Reducing CD8+ T cell growth can stabilize alopecia areata.

γδ T cells are essential for wound healing after poxvirus infection.

The study suggests that a specific type of immune cell, memory-like NK cells, may increase during active hair loss in Alopecia areata.

CD4+ lymphocytopenia can occur in patients with systemic lupus erythematosus.

BST2 protein and certain T cells increase in early alopecia areata.