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Immune checkpoint inhibitors can increase the risk of autoimmune skin diseases, especially bullous pemphigoid.

Cell detachment, not autoantibody binding, causes major changes in pemphigus.

Blocking IL-17 can reduce joint inflammation in Ross River Virus infections.

CD3+ T-cell presence is a reliable marker to tell apart alopecia areata from pattern hair loss.

TH antibodies in vitiligo and AA patients recognize the same protein parts.

Hair loss treatment caused more hair loss in a man.

Catalytic antibodies are early indicators and active participants in the development of systemic lupus erythematosus.

Farudodstat may help prevent hair loss in alopecia areata by stopping immune attacks on hair follicles.

Telitacicept effectively improved hair regrowth in a woman with lupus and alopecia.

Targeting NETs may help reduce fibrosis in Crohn's disease.

IL-4 and IL-13 might play a role in a type of hair loss similar to alopecia areata.

Blocking the CCR5 receptor may be a new way to treat hair loss from alopecia areata.

Alopecia areata shows a unique type 1 interferon signature, suggesting potential treatment by targeting this pathway.

Certain microRNAs may help treat alopecia areata by targeting immune pathways.

Some brain cancer cells avoid immune system detection, and certain treatments could target this to slow their growth; also, certain fat cell precursors help regenerate hair and skin after injury.

ITK inhibitors may effectively treat alopecia areata.

Alopecia areata involves complex immune activity, mainly Th1, with potential benefits from broader immune treatments.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

The study found that a key immune pathway protecting hair follicles is reduced in a mouse model of scarring hair loss.

Alopecia areata involves immune activation in the scalp, suggesting treatments targeting TH1, TH2, and IL-23 pathways.

Targeting specific cell interactions may help treat skin fibrosis.

Corticosteroid therapy reduces specific immune cells and promotes hair growth in alopecia areata patients.

Lymphocytes, a type of immune cell, are crucial for wound healing as they help remodel damaged areas and reduce inflammation.

Abatacept may help some people with alopecia areata regrow hair.

CD271 is crucial for maintaining healthy skin and preventing inflammation.

Anti-Ku-positive patients often have muscle weakness and autophagy plays a key role in their condition.

Etanercept may not prevent alopecia areata from coming back.

The study suggested certain immune cells might cause alopecia areata, but it was retracted.