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Skin grafts from related donors significantly healed chronic wounds in patients with a severe skin condition over a year.

Porokeratotic eccrine and hair follicle nevus is a very rare skin disorder possibly caused by a GJB2 gene mutation.

Microporous scaffolds speed up skin healing and regeneration.

Three specific genetic variants cause severe skin issues in children with EBS, highlighting the need for early genetic screening.

Two fibroblast types shape skin structure and repair differently.

Nipple area expansion in mice needs both pregnancy hormones and mechanical strain.

Mutations in keratin genes cause cell fragility and various skin disorders.

Skin cell behavior is influenced by the tightness of nearby cells, affecting their growth and development.

Tissue-engineered scaffolds help heal difficult wounds by supporting cell growth and repair.

Reductive stress messes up collagen balance and alters cell signaling in human skin cells, which could help treat certain skin diseases.

Cathepsin E is crucial for normal skin cell differentiation and development.

The boy's severe skin disorder is caused by two new mutations in his TGM1 gene.

Epimorphin helps shape and develop epithelial cells, like those in hair follicles.

Specific mutations in a receptor cause facial abnormalities and hair loss.

Mechanical force is important for the first contact between skin cells and hair growth in mini-organs.

The extracellular matrix is crucial for controlling skin stem cell behavior and health.

Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

The stiffness of a wound affects hair growth during healing, with less stiff areas growing more hair.

Researchers fixed gene mutations causing a skin disease in stem cells, which then improved skin grafts in mice.

The newborn's skin blisters healed without scarring, leaving some light spots, and stopped forming after four months.

The enzyme is crucial for skin cell development and can be activated without proteolytic activation.

Three new types of a skin blistering disease were found, caused by specific gene mutations.

Epidermolysis bullosa in calves was not caused by mutations in the keratin genes bKRT5 and bKRT14.

A 37-year-old male with severe skin and internal issues has a rare inherited skin condition called dystrophic epidermolysis bullosa.

Tissue stiffness is influenced by contractility, which suppresses collagen breakdown.

Chemokine signaling is important for hair development.

Disrupting Notch signaling in blood vessels increases scarring during wound healing in mice.

MMP-19 may worsen skin diseases by affecting skin growth and inflammation.

Higher scalp elasticity leads to wider scars after hair transplantation; a new method to measure elasticity may help predict scar size.