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Nail-matrical fibroblasts can make non-nail cells produce hard keratin, useful for nail repair.

Certain genetic variants in ERN1, TACR3, and SPPL2C are linked to when Alzheimer's disease starts.

Correcting EDA fibronectin organization and YAP translocation can improve wound healing in fibrotic conditions.

KSR1 is crucial for certain skin tumor formation and could be a cancer therapy target.

Epimorphin helps shape and develop epithelial cells, like those in hair follicles.

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Eruptive vellus hair cysts likely originate from the infrainfundibulum and sebaceous duct.

The cornea develops independently of the lens, following its own default pathway.

CRISPR-Cas9 allows precise DNA editing but raises ethical concerns about modifying human embryos.

A new keratin, hK6irs1, is found in all layers of the hair follicle's inner root sheath.

GRHL3 is important for controlling gene activity in skin cells during different stages of their development.

Bioengineered lacrimal glands can restore tear production and protect eyes.

CCC1 is essential for ion balance and proper plant cell function.

The committee decided not to approve the drug due to low effectiveness and high risks.

Claudin-1 is crucial for maintaining skin barrier and preventing inflammation.

Serum clusterin may play a key role in the inflammation and immune response in post-adolescent acne.

Crataeva nurvala leaves may help reduce anxiety and improve sleep.

Overexpression of ALK2 in hair follicles disrupts skin development and slows wound healing.

The nanofiber scaffolds improved skin wound healing by supporting cell growth and tissue repair.

Mature hair surfaces are formed by keratinized cells with developed layers, not just modified plasma membranes.

The HCR gene contributes to psoriasis risk.

CRISPR-Cas9 can successfully edit genes in large mammals like Cashmere goats.