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TCDD changes skin cell growth and keratin production in mice.

Dendritic epidermal T cells help wounds heal faster by boosting skin stem cell growth.

Plant-derived amino acids can help develop new antimicrobial drugs.

Alarmin cytokines are key in controlling skin immunity and inflammation.

Some bacteria use arsenic compounds as antibiotics, and others have evolved resistance; a particular arsenic-based compound shows potential as a new antimalarial treatment.

Reptiles on the Absheron Peninsula accumulate toxic elements in their tissues but survive due to tissue resilience.

The enzyme is crucial for skin cell development and can be activated without proteolytic activation.

Type 2 cytokines IL-4 and IL-13 increase skin's vulnerability to Staphylococcus aureus, which could be important for treating atopic dermatitis.

Botulinum toxin type A may widen certain arteries, but more research is needed.

Cepharanthine can block enterovirus entry and protect against infection.

Targeting the TRPV4 channel may help treat intestinal diseases.

Skin bacteria help in skin regeneration and wound healing, with a specific signal called IL-1β playing a crucial role.

Enzymes can release hydrocarbons from Botryococcus braunii without harming cells, suggesting potential for continuous extraction.

A new hydrogel can kill resistant bacteria and help heal infected burn wounds.

Marine compounds from gorgonians and soft corals show promise for drug development, especially for chronic disorders.

Gentamicin-loaded exosomes improve healing of infected diabetic wounds in mice.

People with atopic dermatitis have more CD4+ T cells that respond to a certain bacterial lipid, which may play a role in the skin condition's inflammation.

Mice with damaged skin or hair follicles are more susceptible to anthrax infection.

T cells and bacteria in the gut and skin help maintain health and protect against disease.

Pharmacophore models can predict liver toxicity and central nervous system toxicity, but they have limitations and specific requirements.

Skin bacteria, specifically Staphylococcus aureus, help in wound healing and hair growth by using IL-1β signaling. Using antibiotics on skin wounds can slow down this natural healing process.

Non-canonical Wnt signaling helps intestinal stem cells move to injury sites for tissue repair.

Eosinophilia-myalgia syndrome, linked to contaminated L-tryptophan supplements, caused severe symptoms and some deaths, with long-term effects in survivors.

Toxic Shock Syndrome mainly affects menstruating women, can recur, and is linked to staph bacteria, with rapid treatment being crucial.

PLAU and SerpinB2 affect cell death differently in various forms of leprosy and could be targets for new treatments.

Resident memory T cells and necroptosis may drive fibrosis in eosinophilic fasciitis and morphea.

Low levels of tenuazonic acid can severely damage vital organs.

MAIT cells may help fight COVID-19 but also contribute to severe inflammation.

Euph E and Euri A from Euphorbia neriifolia help control inflammation and immune response in cells.