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Alopecia frontal fibrosante affects facial vellus hair and can be diagnosed with dermatoscopy.

New research suggests that skin cell renewal may not require a special type of cell previously thought to be essential.

Research on epidermal stem cells has advanced significantly, showing promise for improved clinical therapies.

High proliferation and cell delamination drive early skin development, while later stages may not rely on cell division orientation.

Eph/ephrin signaling is important for skin cell behavior and could be targeted to treat skin diseases.

Vitamin D and calcium are important for quick and effective skin wound healing.

Skin health and repair depend on the signals between skin stem cells and their surrounding cells.

Skin stem cells are crucial for maintaining and repairing the skin and hair, using a complex mix of signals to do so.

Stem cells help heal wounds by rapidly dividing and migrating to the wound edge.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

Pregnancy can cause skin pigmentation, stretch marks, and changes in hair, nails, and sweat glands, with most resolving after birth.

Notch1 helps skin heal by attracting specific immune cells.

Keratin 15 is not a reliable sole marker for identifying epidermal stem cells because it's found in various cell types.

The skin protects the body and is constantly renewed by stem cells; disruptions can lead to cancer.

Wnt signaling is essential for forming the skin's spinous layer through a BMP-FGF pathway.

Skin grafting can help repigment vitiligo, but it's less effective for widespread cases due to the Koebner phenomenon.

Skin development in mammals is controlled by key proteins and signals from underlying cells, involving stem cells for maintenance and repair.

Hair follicle regeneration needs special conditions and young cells.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

Toxic epidermal necrolysis is a severe skin condition often caused by drugs, with complex treatment and a high risk of death, but survivors usually heal without scars.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Certain immune system proteins are important for skin healing but can cause problems if there are too many of them.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

Minoxidil promotes hair growth by increasing cell division and DNA synthesis.

Beta-catenin is crucial for skin cell growth, development, and cancer formation.

RANK-RANKL signaling is essential for hair growth and skin health.

Skin stem cells maintain and repair the outer layer of skin, with some types being essential for healing wounds.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

K16 can partially replace K14 but causes hair loss and skin issues.

BLIMP1 is essential for skin maintenance but not for defining sebaceous gland progenitors.