Search

everythinglearnresearchcommunity

for

Search:↓

A new mutation in the STING protein causes a range of symptoms and its severity may be affected by other genetic variations; treatment with a specific inhibitor showed improvement in one patient.

Epithelial-derived Pop-Up Keratinocytes (ePUKs) may enhance wound healing in regenerative medicine.

Tumor cell adhesion is linked to higher risk of SLN metastasis and melanoma recurrence, and a model including these factors predicts these outcomes better than one with just clinical data.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Mouse skin has special cells in the epidermis that decrease with age and are linked to keratinocyte stem cells.

Resident memory T cells and necroptosis may drive fibrosis in eosinophilic fasciitis and morphea.

PROTAC technology shows promise for cancer treatment but needs more effective E3 ligase recruiters.

A new mutation in the STING protein causes a disease with lupus-like symptoms and responds well to a specific inhibitor treatment.

Atopic dermatitis shows a link between skin layers in inflammation, detectable with detailed gene analysis.

The ethosomal delivery system improves topical drug delivery and promotes hair growth.

ELL is crucial for gene transcription related to skin cell growth.

Proteasome inhibitors are promising treatments for various cancers, autoimmune diseases, and other conditions.

Blocking TGFβ-RI signaling enhances surface ectoderm differentiation from human stem cells.

CD8+ T cells attack hair follicle stem cells, causing scarring and hair loss.

Ber‐EP4 marks cells related to the secondary hair germ in hair follicles.

Colorectal cancer's ability to spread is due to changes in many genes, not just one.

ANE syndrome is caused by a mutation in the RBM28 protein that disrupts ribosome assembly.

The growth of the Epstein-Barr virus in the patient's cells was linked to the worsening of her lymphoma.

CXCR2 in skin cells promotes tumor growth.

The research suggests that a specific skin gene can be controlled by signals within and between cells and is wrongly activated in certain skin diseases.

Removing EGFR in skin causes inflammation and abnormal hair growth.

Hair follicles and sweat glands show different keratin staining patterns.

Three molecular subtypes of advanced skin T-cell lymphoma were identified, with potential biomarkers for predicting treatment response and disease progression.

EGF signaling affects gene expression in skin cells, influencing hair growth and potentially cancer.

Antizyme reduces tumor growth and normalizes skin cell development affected by MEK.

The oncoprotein causes abnormal hair growth without increasing skin cancer risk.

Biodegradable scaffolds help regenerate wounds and hair by activating the immune system.

Self-assembling peptide hydrogels effectively deliver drugs locally, enhancing treatment and reducing side effects.