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ERK activation spreads between cells in mouse skin, linked to cell division and influenced by TPA and EGF receptors.

CXCR2 in skin cells promotes tumor growth.

ERK activation spreads between cells, influencing cell division and wound healing.

Lack of KSR1 stops certain skin tumors in mice.

Stem cells control their future role by changing ERK signal timing, affecting tissue regeneration and cancer.

Keratin 15 expression in skin cells is regulated by two mechanisms involving PKC/AP-1 and FOXM1.

XEDAR triggers a specific signaling pathway in cells.

The ID2 gene can help distinguish between sensitizers and irritants in skin cells.

NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

Erlotinib causes skin inflammation through IL-1, which can be reduced by anakinra.

Normal cells outcompete and remove mutant cells in the pancreas with the help of the EphA2 receptor.

Kremen is crucial for proper development and preventing tumors by regulating Wnt signaling.

ACKR2 helps prevent skin scarring and hair loss by controlling inflammation.

EP 2 receptor is essential for heart repair by helping macrophages work properly.

CD271 is crucial for maintaining healthy skin and preventing inflammation.

Blocking EGFR can cause skin inflammation by disrupting IL-1 signaling.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

β1 integrin is essential for the survival, growth, and movement of human epithelial progenitor cells.

Interleukin-1 increases keratin K6 production in skin cells.

Overexpression of E2F1 can lead to skin tumors and disrupt hair growth.

Special gut cells help stem cells move to and fix injured areas by activating a specific signaling pathway.

EdnrB signaling helps melanocyte stem cells regenerate and could be targeted to treat pigmentation issues.

Blocking mTORC1 reduces skin tumor growth in mice.

The gp130 receptor helps in tissue regeneration and disease progression, and manipulating it could improve healing and prevent disease.

Keratin 6A increases skin inflammation, suggesting it could be a target for treating certain skin diseases.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.

Dermal EZH2 controls skin cell development and hair growth in mice.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

Activating cannabinoid receptor 1 reduces certain keratin levels, potentially aiding psoriasis treatment.