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Fadrozole and Finasteride change frog sex ratios and cause intersex animals with altered gene expressions.

miRNAs in the Dlk1-Gtl2 region may improve lamb fur quality.

Very-long-chain acyl-CoA synthetases and fatty acid transport proteins play key roles in fatty acid metabolism and lipid processing in different tissues.

Keratins and filaggrin change as fetal skin develops, marking key stages of skin formation.

Folliculotropic mycosis fungoides has unique molecular features and cell interactions that could guide targeted therapy.

LONRF1 is important for oxidative damage response and tissue remodeling during wound healing.

Knocking out the FGF5 gene in sheep increased wool production and hair-follicle density.

FGF10 and non-coding RNAs are important for cashmere goat hair follicle development.

Disrupting the FGF5 gene in rabbits leads to longer hair by extending the hair growth phase.

A gene mutation in mice causes severe skin disorder similar to a human condition.

FGFR2 signaling controls Merkel cell formation in different skin regions.

FGF-9 speeds up the early development of certain organs, showing potential for organ regeneration.

DNAJB9 cfRNA could help diagnose and treat female hair loss.

SLC3A2 is crucial for hair follicle stem cell function and hair growth.

Fgf20 is important for the development and regulation of the cells that form the base of hair follicles.

The method successfully created stable transfection donor cells for goat hair follicle research.

The research mapped gene activity in developing mouse skin and found key markers for skin cell types and changes from fetal to early postnatal stages.

Editing the FGF5 gene in sheep increases fine wool growth.

The LEF-1 binding site enhances gene expression in hair follicles, with other proteins aiding specific regulation.

Protein profiling of forehead skin can help distinguish between frontal fibrosing alopecia and androgenetic alopecia.

Sheep cells were successfully modified to include a spider silk protein gene.

Frontal fibrosing alopecia is linked to four genetic areas, especially the HLA-B*07:02 allele.

Overexpression of SSAT in mice causes hair loss, liver damage, and sensitivity to polyamine analogues.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Transgenic sheep cells with spider silk gene were successfully created for future sheep hair expression.

D-OCT shows increased blood vessel growth in response to tissue damage in Frontal Fibrosing Alopecia and is useful for diagnosis and monitoring.

Overexpressing follistatin in mice delays wound healing and reduces scar size.

Increased methylation of the Filip1l gene may contribute to aggressive skin cancer.

WNT activation in scalp fibroblasts boosts hair growth by increasing FGF9.

FGF5s can block the effects of FGF5, which may help control hair growth in cashmere goats.