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Methionine supplementation in low-protein diets improves growth, fur quality, and gut health in blue foxes.

Certain genes controlled by OVOL1 are crucial for creating new hair follicles.

About 50% of 46, XY DSD cases lack a genetic diagnosis, but advanced sequencing methods improve detection.

PRP improves hair growth in androgenic alopecia, especially when combined with minoxidil.

CHI3L1 and CXCL5 proteins help promote hair growth.

Hypothyroidism may cause certain types of hair loss.

Male pattern hair loss may be linked to the developmental origins of hair follicles.

Non-coding RNA boosts retinoic acid production and signaling, aiding regeneration.

ROR2 is essential for hair follicle stem cell renewal and maintenance.

Egfl6 is not needed for zebrafish face development.

Folliculin helps regulate energy and nutrient sensing, impacting Birt–Hogg–Dubé syndrome.

TAGX-0003 protected hair follicles and reversed alopecia areata in a mouse model.

Snail1 may contribute to fibrosis in frontal fibrosing alopecia in postmenopausal women.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

miR-140-y helps control feather growth in geese by affecting the Wnt signaling pathway.

Mouse keratin 6 isoforms have different expression patterns in various tissues.

Adenosine may promote hair growth by increasing FGF-7 levels in dermal papilla cells.

EBF1 controls hair type and length.

Blocking LFA-1 prevents hair loss in mice.

Mutations in the DSG4 gene can cause a rare hair disorder similar to monilethrix.

Farudodstat can prevent hair follicle immune damage linked to alopecia areata.

Cells in hair follicles help create fat cells in the skin by releasing a protein called Sonic Hedgehog.

VYN201 shows promise as a safe and effective treatment for non-segmental vitiligo.

Lower TGFß1 levels help stem cells become beige fat cells.

EGF–FGF2 helps mouse stem cells grow and become more like nerve cells.

BBS7 is crucial for maintaining healthy periodontal ligaments by supporting Shh signaling.

AP-1 and TGFß work together to drive resistance in basal cell carcinoma, suggesting new treatment options.