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Hereditary hyperplastic gingivitis in silver foxes may be linked to errors in the MAPK signaling pathway, influenced by androgens.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

RXRa is crucial for Th2 immune cell development and may link nutrition to immune health.

A new PAX9 gene mutation causes missing teeth and hair problems, but not skin or nail issues.

Removing Sprouty genes in mice causes various hormone-related issues but does not increase cancer risk by one year of age.

The document concludes that MGRN1 affects mouse fur color by interfering with a receptor's signaling, but its full role in the body is still unknown.

Pax9 is crucial for proper tongue surface development and preventing skin-like changes.

Mutations in the POC1A gene can cause a unique form of extreme insulin resistance and short stature.

LONRF1 is important for oxidative damage response and tissue remodeling during wound healing.

Lack of KSR1 stops certain skin tumors in mice.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Fox Fordyce disease might be more common in prepubertal girls than thought and can be managed with treatment.

Mutations in the SHH pathway in certain skin cells can cause skin tumors and abnormal hair growth.

Mutations in β1 integrins cause embryonic death but have milder effects on skin.

Introducing the rat OTC gene partially corrected OTC deficiency in mice.

Deleting the CDSN gene causes severe skin and hair problems, leading to death.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

Removing both KLK5 and KLK7 proteins can prevent death and skin issues in Netherton syndrome.

A missing mK6irs1 gene causes hair loss in mice.

Blocking LFA-1 prevents hair loss in mice.

Mice without certain skin proteins had abnormal skin and hair development.

Minoxidil treatment improves heart defects in a DiGeorge syndrome model.

Nude mice are hairless due to a shared defect affecting both skin and thymus, not just thymic issues.

A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

A new rat strain with a specific gene mutation causes hair loss and kidney issues.

Runx1 controls fat-related genes important for normal and cancer cell growth, affecting skin and hair cell behavior.

Mice without the Sp6 gene have problems developing several body parts, including hair, teeth, limbs, and lungs.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.