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TTD hair is brittle due to fewer sulfur amino acids and unstable disulfide bonds.

Four children had unmanageable pale blond hair due to uncombable-hair syndrome.

The hair follicle structure is more complex than thought, with new findings on protein formation.

Cosmetic products or emotional factors might contribute to Frontal Fibrosing Alopecia, and trichoscopy is useful for diagnosis.

Hair treatments like bleaching increase friction by exposing tiny pores on the hair surface.

Being allergic to linalool, a common fragrance ingredient, might contribute to developing frontal fibrosing alopecia.

A mutation in the FAM83G gene is linked to skin and hair abnormalities in two related individuals.

Mutations in the hairless protein gene cause hair loss.

Human hair has different protein structures in its cuticle and cortex.

Hair movement can indicate hair quality and health.

Frontal fibrosing alopecia is a scarring hair loss condition mainly affecting postmenopausal women, with unclear causes.

Mouse profilaggrin helps in skin cell differentiation and may be involved in calcium signaling.

Woman has discoid lupus, frontal fibrosing, and androgenetic alopecia.

A specific mutation in PA-PLA1α causes abnormal hair growth.

Deleting part of a gene in mice causes wavy hair and high pup loss.

The patient's hair has unique structural differences with alternating bright and dark bands.

Cosmetic treatments can replenish key amino acids in damaged hair, improving its strength and appearance.

Using facial sunscreen and moisturizer may increase the risk of frontal fibrosing alopecia.

Disulfide bonds affect the melting behavior of hair's crystalline structure, but hair retains some stability even after these bonds are broken.

Linalool in fragrances may harm hair follicles and contribute to hair loss.

Certain drugs and supplements may contribute to hair loss in the frontal hairline in older women.

Clim proteins are essential for maintaining healthy corneas and hair follicles.

Claudin-1 is important for the barrier function and growth of hair.

Frontal fibrosing alopecia causes hair loss at the front hairline, and no effective treatment exists.

Frontal fibrosing alopecia may be caused by an autoimmune reaction and hormonal imbalance.

Frontal fibrosing alopecia is a poorly understood condition with increasing cases and unclear treatment effectiveness.

Mutations in the DSG4 gene cause fragile, sparse hair in humans, mice, and rats.

The Glu413Lys mutation in keratin affects hair stability, while Glu413Asp does not.

Hair loss in male frontal fibrosing alopecia may spare areas covered by a wristwatch.