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Circular RNA ERCC6 helps activate stem cells important for cashmere goat hair growth by interacting with specific molecules in an m6A modification-dependent way.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.

LEF-1 boosts cell growth in goat hair follicles, aiding cashmere production.

Keratinocytes can reduce the survival of certain melanoma cells, suggesting new therapy paths.

Dermal EZH2 controls skin cell development and hair growth in mice.

AP-1 and TGFß work together to drive resistance in basal cell carcinoma, suggesting new treatment options.

CLE40 and CRN/CLV2 pathways have opposite effects on root growth in Arabidopsis.

Truncated LTBP-1 disrupts TGF-β signaling, affecting hair growth.

Keratinocyte growth factor significantly alters skin and tissue development.

Sweat gland stem cells help maintain glands, aid wound healing, and can regenerate skin structures.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

Kremen is crucial for proper development and preventing tumors by regulating Wnt signaling.

ELL is crucial for gene transcription related to skin cell growth.

The E2 protein affects gene activity in hair follicles of mice.

A small group of slow-growing cells causes basal cell carcinoma to return after treatment.

CTCF protein is essential for skin and hair follicle development in mice.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

CENPV, a new partner of CYLD, helps regulate ciliary acetylated tubulin and is overexpressed in certain skin tumors.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

Calretinin identifies the companion cell layer in human hair follicles.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Nuclear Factor I-C is important for controlling hair growth by affecting the TGF-β1 pathway.

LGR5 and LGR6 are expressed differently in various skin tumors, which may offer clues about their origins.

Scientists made a mouse that shows how a specific protein in the skin changes and affects hair growth and shape.

Inactivating β-catenin is essential for chick retina regeneration.

CRISPR/Cas9 and prime editing can potentially fix skin disorder genes safely and effectively.

Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

Delta1 is crucial for controlling skin cell growth and preventing tumors in mice.

pCLCA2 protein may help maintain skin structure and function.