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A 4kb fragment of the desmocollin 3 promoter targets gene expression to specific skin and hair follicle areas.

A new method allows for controlled, long-lasting delivery of retinoic acid through the skin with less inflammation.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Certain proteins and their receptors are more active during the growth phase of human hair and could be targeted to treat hair disorders.

Light can turn on hair growth cells through a nerve path starting in the eyes.

Retinoids or their analogs could treat skin pigmentation disorders like melasma and vitiligo.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

The structure of SRD5A reveals how it reduces steroids, aiding drug design for related health conditions.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

PPAR-γ may be a key target for treating alopecia areata and other skin conditions.

A substance called poly(I:C) increases a protein called carbonic anhydrase II in skin cells, which might help with skin defense and healing.

A new method accurately measures a drug in skin for cancer therapy research.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

Tyrosinase and gp100 proteins can help diagnose and treat melanoma.

Different types of human skin cells respond uniquely to various colors and doses of light, which could lead to specialized light treatments for skin conditions.

TRPV4 helps sense pressure in mouse skin.

ABCG2 protein marks stem-like skin cells in human epidermis.

LHX2, with other markers, can identify hair placodes in rats.

CAP1/Prss8 does not activate PAR2 or inhibit PN-1.

The document confirms the structures of major metabolites of the CRTh2 antagonist Setipiprant and identifies minor metabolites.

KRT14 gene variants cause dermatopathia pigmentosa reticularis, affecting nails, teeth, and hair.

Human skin's melanocytes respond to light by changing shape, producing pigments and hormones, which may affect sleep patterns.

Different light affects cell functions and can help treat skin conditions.

Rapp-Hodgkin syndrome, AEC, and EEC are different expressions of the same genetic disorder caused by TP63 gene mutations.

PRX01, PRX44, and PRX73 are essential for root hair growth in Arabidopsis thaliana.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

The glucocorticoid receptor helps protect skin from tumors and other issues.

Rhino mice show significant meibomian gland changes, making them a potential model for studying gland disorders.