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Beige and leaden pigment genes act within melanocytes, affecting pigment patterns.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.

Gingerol may help treat chronic graft-versus-host disease by improving immune cell balance.

Mice with human-like EGFR had growth issues, skin defects, heart problems, and unusual bone development.

Removing GRK2 in skin cells causes hair loss similar to immune-related alopecia.

Exposure to low doses of finasteride in the womb causes changes in the prostate of both male and female gerbils.

The glucocorticoid receptor helps protect skin from tumors and other issues.

The frizzy mouse and hairless rat mutations are due to changes in the Prss8 gene.

SMR-2 and SMR-6 are much more toxic than retinoic acid, causing severe symptoms and organ damage in mice.

The BMP2 gene is more active in the early growth phase of Cashmere goat hair and may affect hair regeneration and textile production.

Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

Activating the GDNF-GFRα1-RET signaling pathway could potentially promote skin and limb regeneration in humans and could be used to treat hair loss and promote wound healing.

Sonic hedgehog signaling is needed for the development of touch-receptor cells in the skin, and the loss of Polycomb repressive complex 2 can lead to more of these cells.

Hedgehog signaling is crucial for mammary gland development over hair follicles.

A new Wnt surrogate specifically targets the Frizzled7 receptor, promoting organoid formation and hair growth.

Mice that can regenerate tissue have cells that pause in the cell cycle, which is important for healing, similar to axolotls.

Mutations in iRhom2 affect hair and skin in mice and are linked to esophageal cancer, with ADAM17 playing a crucial role.

Slug (Snai2) helps regulate hair growth timing in mice.

Agouti protein affects melanocortin receptors through competitive antagonism and receptor down-regulation.

LGR5 mainly stays inside cells, moving to the trans-Golgi network, and this process is important for its role in cell signaling.

Overexpressing the epigen gene in mice leads to enlarged sebaceous glands and greasy fur.

Retinoic acid causes gland formation instead of hair in mouse skin by altering epidermal and dermal interactions.

Rhino mice show significant meibomian gland changes, making them a potential model for studying gland disorders.

Researchers created a new mouse model, G4, that mimics human PCOS symptoms and links the condition to a specific gene.

Dermal EZH2 controls skin cell growth and differentiation in mice.

The E2 protein affects gene activity in hair follicles of mice.

The mineralocorticoid receptor temporarily affects mouse skin development, but the glucocorticoid receptor has a more lasting impact.

EGFR helps mouse hair follicles stop growing by reducing certain growth regulators.

Mice skin has components that could help with hair growth and might be used for diabetes treatment.