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Early involution in Hirosaki hairless rats' mammary glands is linked to a unique modification of STAT5A.

Hairless protein can block vitamin D activation in skin cells.

TGase 3 helps build hair structure by forming strong bonds between proteins.

61 functional keratin genes were identified in dogs and horses, leading to updated gene annotations.

Keratin proteins in hair are complex and come from multiple gene families.

c-Myc, Max, and Bin1 help hair follicle cells mature and die.

AgK114 protein helps in hamster skin injury recovery.

Keratin 79 cells help form and regenerate hair canals.

The protein p53 directly reduces the production of Keratin 17, a skin and hair protein, in rats with radiation dermatitis.

Scientists found new and known long non-coding RNAs in mouse hair follicle stem cells that may be important for stem cell function and could be targets for cancer treatment.

Ptprq has multiple forms that change during inner ear development.

A new genetic region, 17q21.31, is linked to higher ovarian cancer risk.

Hedgehog signaling is essential for normal sebaceous gland development and affects keratin 6a expression.

A special receptor in sensory nerve endings helps control how they respond to stretching.

A mutation in the human hairless gene causes alopecia universalis.

Artemis protein may help control hair growth and health by influencing cell processes.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Polyamines help fix DNA damage accurately in cells.

Key skin cell regulators and gene organization changes are crucial for skin cell development and could help treat skin disorders.

Two new mutations in the CDH3 gene cause hair loss and vision problems in a young girl.

SGK3 is essential for proper hair growth and health.

Two new gene mutations cause a rare hair disorder.

Changing YBX1 protein activity affects skin stem cell function and aging.

The mutation causes hairless mice due to mislocalized and dysfunctional HR protein.

Sox9 is crucial for hair follicle stem cells to become melanocytes instead of glial cells.

Keratin K14 expression varies between hair follicles and epidermis, affecting cell differentiation.

Antizyme reduces tumor growth and normalizes skin cell development affected by MEK.

EBV infection increases a specific keratin variant in carcinoma cells, possibly affecting cell structure and cancer progression.

Key genes linked to hair growth and cancer were identified in hairless mice.

FOXN1 duplication can cause excessive hair growth.