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Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

Pdgfα signaling helps maintain fat cells in the skin and is important for hair growth, wound healing, and fighting infections.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Hair greying is caused by oxidative stress damaging hair follicles and melanocytes.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

Deleting podoplanin in mice promotes hair growth by enhancing cell migration.

Special immune cells called Treg cells are important for maintaining and regenerating hair by activating a specific growth signal in hair stem cells.

Deleting the CRIF1 gene in mice disrupts skin and hair formation, certain proteins affect hair growth, a new compound may improve skin and hair health, blood cell-derived stem cells can create skin-like structures, and hair follicle stem cells come from embryonic cells needing specific signals for development.

MED1 is essential for normal hair growth and maintaining hair follicle stem cells.

Lack of Crif1 in hair follicle stem cells slows down hair growth in mice.

Aging changes hair stem cells and their environment, leading to gray hair and hair thinning, but understanding these changes could help develop treatments for hair regeneration.

The study found new details about human hair growth and suggests that preventing a specific biological pathway could potentially treat hair graying.

Different problems with hair stem cell renewal can lead to hair loss.

PIEZO1 helps keep hair follicle stem cells inactive, affecting hair growth.

The Msi2 protein helps keep hair follicle stem cells inactive, controlling hair growth and regeneration.

Nestin marks cells that can become a specific type of skin cell in hair follicles of both developing and adult mice.

Stress can cause hair to turn gray by depleting pigment-producing cells through the release of a stress hormone.

Traction may not be the only cause of cicatricial marginal alopecia.

Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

Cancer treatment drugs can cause permanent hair loss by damaging hair follicle stem cells, but a specific inhibitor might reverse this effect.

TACE/ADAM17 is essential for maintaining healthy hair and hair follicle stem cells.

Stress can cause hair loss by affecting nerve-related hair growth, and noradrenaline might help prevent this.

The nervous system helps control stem cell behavior and immune responses, affecting tissue repair and maintenance.

GRK2 is essential for healthy hair follicle function, and its absence can lead to hair loss and cysts.

Inactive hair follicle stem cells help prevent skin cancer.

Dying cells can help with faster healing and new hair growth by releasing a growth-promoting molecule.

Stem cell behavior varies with stimuli, and lineage changes can happen without affecting stem cell division.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Cells lacking the Bax protein can outcompete others, leading to better tissue repair and hair growth.

Compartmentalized organization might be crucial for stem cells to effectively respond to growth or injury.