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Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

Keratinocyte growth factor significantly alters skin and tissue development.

Ectodysplasin-A1 is crucial for developing hair, teeth, and glands.

The Foxn1(-/-) phenotype disrupts hair growth and affects skin stem cells.

Introducing the rat OTC gene partially corrected OTC deficiency in mice.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

Msx2 deficiency in mice leads to bone growth and organ development problems.

The method successfully isolates hair follicle stem cells from mice for research.

Human placenta hydrolysate reduces inflammatory pain and nerve damage in mice.

Mus pahari mice have fragile skin due to abnormal collagen and elastin.

Deleting Snai2 and Snai3 causes fatal autoimmunity.

TAGX-0003 protected hair follicles and reversed alopecia areata in a mouse model.

Msx-2 gene removal speeds up skin wound healing in mice.

Mice with enhanced regeneration abilities may help develop new regenerative medicine therapies.

Increased ornithine decarboxylase makes normally tumor-resistant mice more sensitive to tumors.

A new mouse gene, Keratin 17n, is mainly found in nail tissue and may explain why mice without Keratin 17 don't have nail issues.

Ephrins slow down skin and hair follicle cell growth.

Mice healed without scars as fetuses but developed scars as adults, suggesting scarless healing might be replicated with further research.

Rat hair-follicle stem cells can become heart cells with specific supplements.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

Mice with too much Claudin-6 have skin barrier problems and abnormal hair growth.

Disrupting Notch signaling in blood vessels increases scarring during wound healing in mice.

Deleting podoplanin in mice promotes hair growth by enhancing cell migration.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

The vector successfully directed specific gene expression in hair follicles.

The Foxn1 gene is essential for normal nail and hair development.

Mice without the IL-6 gene had more hair growth after injury due to higher activity of a related protein, Stat3.