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Electrospun gelatin-based nanofiber dressings are promising for wound healing due to their effective healing properties and ability to protect against infections.

Researchers developed new compounds that target the androgen receptor effectively with fewer side effects.

Large-scale fibronectin nanofibers help heal wounds and repair tissue in a skin model of a mouse.

MDSC-Exo can treat autoimmune alopecia areata and promote hair regrowth in mice.

Vitamin D3 is important for bone health and may help treat various diseases beyond bone-related conditions.

Vitamin D3 affects cell differentiation in specific skin areas.

The enzyme for activating vitamin D is found in many body tissues, not just the kidneys.

Small amounts of Vitamin D3 can boost hair growth, but too much can stop it.

Measuring 24-OHase induction helps identify defects in vitamin D processing and predict treatment response.

DDR-II hair follicle cells don't respond to 1, 25-dihydroxyvitamin D3, aiding diagnosis.

A specific gene mutation causes vitamin D resistance, but certain vitamin D analogs might help.

Alopecia indicates more severe resistance to 1,25-dihydroxyvitamin D.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requires high doses of calcium and vitamin D for management.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

Patients with this syndrome can have different responses and worsening resistance to treatment over time.

A genetic defect in vitamin D receptors causes severe rickets and hair loss in children, but some heal as they age.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

1α,25-dihydroxyvitamin D3 directly affects cartilage growth and development.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Child with rickets improved with a specific vitamin D therapy, but alopecia did not change.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

Estrogen deficiency can reduce the enzyme activity needed to activate vitamin D.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

1,25-(OH)2D3 helps hair grow by blocking certain harmful signals.

The vitamin D receptor can work without its usual activating molecule.

Rubbing vitamin D3 on skin can help prevent hair loss from chemotherapy and slow breast tumor growth in mice.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

A vitamin D receptor mutation causes rickets and affects immune responses.

New vitamin D3 forms need the vitamin D receptor to reduce fibrosis in human cells.