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Male mice are more susceptible to autism-like changes from valproic acid than female mice.

5-alpha reductase inhibitors, like finasteride and dutasteride, may cause depression, but more research is needed to understand why.

Altered metabolism can help control seizures by changing brain signaling and energy use, suggesting new treatments for epilepsy.

Acetate helps reduce depression in rats with PCOS by lowering specific gene expression and DNA changes in the brain.

Human hair follicle stem cells improved memory and brain health in rats.

Finasteride affects the male rat brain by reducing certain protein activation, but these effects may reverse after stopping the drug.

Social isolation changes brain receptors and makes ethanol more impactful.

Osteopathic manipulation therapy might help treat dementia caused by finasteride.

Short-term finasteride use in male rats caused anxiety, depression, and memory problems.

Hair follicles supply a crucial brain development protein to the brain via platelets.

Finasteride may reduce depression and anxiety in female rats and improve brain connectivity.

Changes in early neurosteroid levels can affect adult learning and anxiety.

Progesterone helps adult male mice's brain cells survive and improves learning and memory.

p53 protein may help protect or kill neurons under stress.

Trilostane may help delay epilepsy development by increasing certain brain chemicals.

TSPO might help treat anxiety and depression.

Long-term alcohol exposure alters brain receptor function differently in various brain regions.

Neurosteroid production in the brain may delay seizure onset.

Letrozole reduces seizures but not brain damage in mice.

Anabolic-androgenic steroids can damage the brain, causing mood and cognitive problems.

Chronological age affects the mouse auditory cortex more than hearing loss, and the brain may be less sensitive to mitochondrial dysfunction.

Short-term diabetes can cause changes in brain steroids, cholesterol balance, and mitochondrial function.

Midazolam's antiseizure effects are mainly due to synaptic GABA-A receptors, not neurosteroids or extrasynaptic receptors.

2-DG reduces seizures by enhancing brain inhibition through specific receptor activation.

Progesterone affects GABA_A receptor function by altering δ subunit levels.

Neurosteroids from glia cells help control seizure development in epilepsy.

Neurosteroids may help prevent seizures in epilepsy.

Neurosteroids like allopregnanolone help control cell death and growth in the developing fetal brain.

Neurosteroids help reduce seizures, but their withdrawal increases seizure activity.

The microneedle system shows promise for non-invasive brain drug delivery.