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A new computer tool quickly measures hair thickness differences in people with common types of hair loss.

The connective tissue around hair follicles changes structure throughout the hair cycle.

Amelanotic melanocytes from hair follicles are immature and likely don't transfer melanosomes to keratinocytes.

Hedgehog signaling controls hair follicle development and can affect skin cancer growth.

Root hairs help barley grow better in zinc-deficient soil.

Fibroblast spheres can form stem cells, but marker distribution needs more study.

Hirsutism is common body hair growth due to genetics and hormones, and while not a disease, it can be distressing; virilisation includes hirsutism with other male traits and needs medical attention.

The hairless mutant gene causes early hair loss and affects skin and thymus development in mice.

The hairless gene mutation causes baldness by disrupting hair follicle structure.

Different miRNAs in Rex rabbit skin affect cell processes and hair growth.

Melanocyte precursors in human fetal skin follow a specific migration pattern and some remain in the skin's deeper layers.

The enzymes Tet1, Tet2, and Tet3 are important for the development of hair follicles and determining hair shape by controlling hair keratin genes.

HPV16-transformed cells can change human skin cell properties, aiding tumor growth.

Rabbits lacking the Hoxc13 gene show similar hair and skin issues to humans with ECTD-9, making them good for research on this condition.

The study found that different genes are active in cashmere goats' hair growth stages, which can help improve cashmere production.

Different types of dog hair loss are linked to problems starting the hair growth phase and early hair cycle ending.

Hair follicle cells become four types: medulla, cortex, cuticle, and inner root sheath.

Hair growth is influenced by various body and external factors, and neighboring hairs communicate to synchronize regeneration.

Researchers developed a method to create artificial hair follicles that may help with hair loss treatment and research.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

Hairlessness in mammals is due to complex genetic changes in both genes and regulatory regions.

AGA and TE can be differentiated by specific trichoscopic features, with AGA showing more variation in hair thickness in fronto-temporal areas.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.

THH and PAD type III appear together early in hair growth, but deiminated THH appears later, suggesting another factor is involved.

Certain signals are important for reducing specific chemical markers on hair follicle stem cells during rest periods, which is necessary for healthy hair growth.

Hairless protein reduces Msx2 gene activity, affecting hair follicle development.

Telocytes in silky fowl embryos develop distinct features and connections by the 20th day of incubation.