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Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

Gene variation affects prostate issues and hair loss.

Hyaluronan from Has2 is important for proper wound healing and hair follicle development.

CARASIL, a rare genetic disorder, was confirmed in an Arabic woman, highlighting its rarity and need for stroke prevention.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

A new mutation in the ST14 gene was found in a patient with ARIH syndrome, showing milder symptoms and no tooth issues.

Vitamin D receptor helps prevent skin cancer and supports skin health.

Certain immune cells may cause hair loss by reacting to stressed hair follicles.

Higher levels of IL-1R1 and hsa-miR-19b-3p may help diagnose and predict alopecia areata severity.

The humanized AA mouse model is better for testing new alopecia areata treatments.

Certain genetic variants increase the risk of resistance to hormone therapy in prostate cancer patients.

The E2 protein affects gene activity in hair follicles of mice.

Different γδ T cell types have unique roles in causing alopecia areata.

A new genetic mutation in the ODC1 gene causes developmental delay and other symptoms in a young girl.

The SHJH hr mice with a mutated Hr gene show signs of faster skin aging due to poor antioxidative protection.

Lower 2D:4D finger ratios in HS patients suggest prenatal hormone influence on the disease.

Vitamin D receptor is essential for hair growth and preventing hair loss.

Atopic dermatitis increases the risk of some autoimmune diseases.

A genetic defect causes males in some Mediterranean populations to be born with ambiguous genitalia and develop male traits at puberty.

Finger length ratios don't predict baldness in men.

Mice with a changed Hr gene lose and regrow hair due to changes in the gene's activity.

A protein called PCBP2 controls the production of a hair growth protein by interacting with its genetic message and is linked to hair loss when this control is disrupted.

A person with a specific gene mutation had extra teeth, unique jaw and hair features not seen before in this condition.

Lack of vitamin D receptor causes hair loss in mice by allowing certain genes to overactivate.

A genetic variant in the androgen receptor gene increases heart disease risk in women but not in men.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

The visfatin GT genotype may increase the risk of Alopecia Areata.

MC4R gene variants not linked to female hair loss.

A new gene location for Keratosis follicularis squamosa was found on chromosome 7p14.3-7p12.1.

A gene mutation in Lama3 is linked to a common type of hair loss.