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Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

Hairless protein can block vitamin D activation in skin cells.

Certain gene patterns in breast cancer are linked to how active hormone receptors are and could affect patient survival.

Hairless protein and putrescine regulate each other, affecting hair growth and skin balance.

CDK4 levels affect the number of hair follicle stem cells in mice.

PBX1 helps hair stem cells grow and change by turning on certain cell signals and preventing cell death, which may be useful for hair regrowth treatments.

Only one of the two K16 genes on chromosome 17 makes a functional protein for keratin filaments.

Chemotherapy damages hair follicles, causing hair loss and other cellular changes.

Certain mutations in a hair growth-related gene cause a type of genetic hair loss.

Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

Bcl-2 helps hair regeneration but can also increase cancer risk.

Human hair keratin genes are similar to mouse genes and are specifically expressed in hair follicles.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

Chemotherapy and radiation therapy cause skin and hair damage by altering gene expression and signaling pathways.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive forestomach cancer.

SP1 promotes and KROX20 inhibits hair cell growth by affecting the CUX1 gene.

Genetic testing for hairless gene mutations is crucial to correctly diagnose and treat atrichia with papular lesions.

The analysis of a large pilomatricoma revealed five distinct areas with different gene activity related to hair growth and tumor development.

The document concludes that the human keratin gene cluster is complex, with a need for updated naming to reflect over 50 functional genes important for hair and skin biology.

Genetic mutations cause various hair diseases, and whole genome sequencing may reveal more about these conditions.

Substances from human hair cells can affect hair loss-related genes, potentially leading to new treatments for baldness.

Researchers found a new mutation in the HR gene linked to a rare hair loss condition.

Targeting Keratin 17 may help overcome cancer therapy resistance.

Human hair keratin genes hHa2 and hHb1 are located on chromosomes 17 and 12.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.

Too much parathyroid hormone-related protein in skin disrupts hair growth in mice.

A specific mutation known for causing cancer is also found to cause a skin condition in a young girl.

Hair follicles have a more inactive cell cycle than other skin cells, which may help develop targeted therapies for skin diseases and cancer.