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Urotensin II increases growth and VEGF production in rat skin cells by turning on the Wnt-β-catenin pathway.

Endothelial TLR2 is crucial for timely wound healing, but HFSC TLR2 is not needed.

The mystacial pad's innervation in adult rats is more complex than previously thought.

Mutations in the SNRPE gene cause hereditary hair loss.

SRD5A1 is crucial in advanced prostate cancer, and blocking both SRD5A1 and SRD5A2 is more effective than targeting SRD5A2 alone.

Too much parathyroid hormone-related protein in skin disrupts hair growth in mice.

Trps1 is essential for proper hair follicle development.

A specific ATR gene mutation is linked to a hereditary oropharyngeal cancer syndrome.

Hsc70 protein may influence hair growth by responding to androgens.

TIP39 and PTH2R help control calcium levels and skin cell development.

FTase and GGTase-I are essential for skin keratinocyte health.

Human hair follicles have a scent receptor that can influence hair growth.

A new gene mutation is linked to monilethrix in the studied family.

A new syndrome was linked to two new genetic changes in the MBTPS1 gene in a 14-year-old girl.

Type 3 5α-reductase is more common and finasteride and dutasteride strongly inhibit it.

Sebocytes play a key role in controlling androgen levels in human skin.

Women with excessive hair growth or polycystic ovary disease may more often carry a gene variant for 21 hydroxylase deficiency.

The hairless protein is important for skin, hair, and may influence cancer development.

Two distinct caspases in human skin help with cell death and skin formation.

Targeting METTL1 may help slow papillary thyroid cancer growth and spread.

Targeting Keratin 17 may help overcome cancer therapy resistance.

The Thr1022Ala variant in the hairless gene is not a disease-causing mutation.

A gene mutation causes curly hair and hair loss in rats.

Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

CRH can cause hair loss by promoting cell death in hair growth cells.

Type II keratins are uniquely phosphorylated during stress and mitosis, affecting their structure and function.

Three specific mutations in the LIPH gene can cause hair loss by damaging the protein's structure and function.

Targeted protein degraders show promise in treating cancer but need to target more diverse proteins.

Autoimmune reactions may cause both alopecia areata and HAM.

A mutation in the Soat1 gene causes hair structure defects and other health issues in AKR/J mice.