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Hair germ cells differ from epidermal cells in keratin expression, and specific keratins form after hair differentiation.

High levels of HSP70 and IL-15 are linked to more severe alopecia areata.

Hemoglobin α acts as an antioxidant in skin cells, especially protecting hair follicle stem cells from UV damage.

CD28 is a promising target for treating alopecia areata with belatacept.

CD28 is a promising target for treating alopecia areata with belatacept.

IL-17 is more important than IFN-γ in causing severe hair loss in chronic alopecia areata.

Malondialdehyde-modified DNA may trigger an immune response in alopecia areata patients.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

Inactive stem cells in hair follicles and muscles can avoid detection by the immune system.

Transplanted hair follicles can change and adapt to new areas of the body, with the immune system possibly playing a role in this adjustment.

Candida auris uses the immune response to colonize hair follicles.

Long COVID patients may have hormonal imbalances linked to their symptoms.

Alopecia areata involves immune activation in the scalp, suggesting treatments targeting TH1, TH2, and IL-23 pathways.

Ovalbumin–aluminum sensitization causes increased pain sensitivity and nerve changes in mice.

Restoring hair follicle immune privilege may help treat alopecia areata.

Hyaluronidase speeds up wound healing and reduces inflammation.

BST2 protein and certain T cells increase in early alopecia areata.

Plasmatherapy improves immune status in vitiligo patients, especially those with segmental vitiligo.

People with alopecia areata often have abnormal thyroid hormones, thyroid antibodies, antinuclear antibodies, low vitamin D, and high C-reactive protein levels.

Sebaceous glands play a key role in skin health, immunity, and various skin diseases.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

Enhancing regulatory T cells may help treat autoimmune diseases like alopecia areata.

The study found that a one-step antibody method is better than the LSAB method for accurately studying hair follicle structures without false positives.

CD8+ T cells attack hair follicle stem cells, causing scarring and hair loss.

Tph cells are linked to the severity of systemic lupus erythematosus.

Catalytic antibodies are early indicators and active participants in the development of systemic lupus erythematosus.

CAR-T cell therapy shows promise for treating autoimmune disorders but faces challenges like complex manufacturing and limited tissue penetration.

People with alopecia areata have fewer IL-10 producing immune cells, which might contribute to the condition.

Hair follicles help attract immune cells to minor skin injuries.