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Twist1 helps maintain important features of cells crucial for hair growth by working with Tcf4 and β-catenin.

H3K4me3 helps control RSPO3 to influence hair growth and development.

The BMP/Smads pathway and Id2 gene control hair follicle stem cells, affecting their rest and growth phases.

Two distinct caspases in human skin help with cell death and skin formation.

Mettl3 is essential for normal tissue development and self-renewal by regulating gene expression.

AP-1 controls tumor cell type by affecting key signaling pathways.

ceRNA networks offer potential treatments for skin aging and wound healing.

N-WASP is essential for normal hair growth in mice.

KGF-1 135 is a stable and effective alternative for treating oral mucositis.

Proteolytic enzymes damaged hair follicle stem cells in transgenic mice.

TACE/ADAM17 is essential for maintaining healthy hair and hair follicle stem cells.

YH0618 helps reduce chemotherapy-induced hair loss by targeting specific proteins and pathways.

A new DSG4 gene mutation causes hair defects in a young girl.

Arabidopsis Formin 2 stabilizes actin filaments, affecting cell-to-cell movement and virus susceptibility.

Type XIX Collagen is present in specific skin and hair cells during development.

Notch1 signaling is impaired in hidradenitis suppurativa, affecting skin and hair cells.

Activin A and follistatin control when hair cells develop in mouse ears.

HOXC13 is essential for hair and nail development by regulating Foxn1.

The 3D scaffold helped maintain hair cell traits and could improve hair loss treatments.

The gelatin/β-TCP scaffold with nanoparticles improves wound healing and skin regeneration.

Finasteride may help treat Neutrophil Actin Dysfunction by reducing LSP1 gene activity.

BcFLA1 protein is crucial for root hair growth in response to low phosphate in Brassica carinata.

The PML protein helps prevent skin cancer in mice.

Arginine converts to citrulline in hair follicles as proteins harden.

A gene called APCDD1, which controls hair growth, is found to be faulty in a type of hair loss called hereditary hypotrichosis simplex.

Tissue-engineered skin substitutes can model junctional epidermolysis bullosa and may help develop gene therapy.

Mutations at Val-889 and Arg-752 disrupt key interactions in the androgen receptor, affecting its function.