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A new treatment using a special gel with miR-665 reduces inflammation and helps hair regrow in alopecia areata.

Faulty inflammasome activation may lead to autoimmune skin diseases and could be a target for new treatments.

Personalized thiopurine dosing based on NUDT15 genotyping can improve long-term outcomes for ulcerative colitis and Crohn's disease patients.

Certain RNAs may help diagnose alopecia areata by affecting keratin genes.

Immune cells and plasma proteins are linked to hair loss, suggesting new treatment options.

These specific gene polymorphisms are not linked to Alopecia Areata in Egyptians.

Alopecia areata shows a unique type 1 interferon signature, suggesting potential treatment by targeting this pathway.

The LEF-1 binding site enhances gene expression in hair follicles, with other proteins aiding specific regulation.

Higher levels of CCL7 are linked to more severe hair loss in alopecia areata patients.

Adjusting the medication tacrolimus resolved a boy's red nail beds after a stem cell transplant.

Careful monitoring is crucial to prevent JC virus reactivation in lupus patients treated with rituximab.

IL-17 inhibitors for psoriasis may cause unexpected hair loss.

Activating liver X receptors can reduce fibrosis by stopping certain immune cells from releasing harmful proteins.

People with alopecia areata have higher levels of RBP4 protein and antibodies against it.

Key genes linked to immune response are highly active in lupus-affected hair follicles.

Targeting immune pathways like JAK/STAT may help treat frontal fibrosing alopecia.

Monocyte-derived dendritic cells play a key role in UVB-induced skin sensitivity and inflammation.

The role of γδT-cells in causing alopecia areata remains unclear.

Controlling Tslp can improve health in AEC syndrome patients.

CD98hc's role in skin health decreases with age.

A new TP63 mutation was found in a baby with EEC syndrome, showing the need for TREC testing to check for immune issues.

A new diagnostic model can help better diagnose and understand Alopecia Areata.

AL136131.3 slows hair growth by affecting energy processes in hair loss.

Higher activity in lichen planopilaris is linked to certain immune and tissue genes.

Alopecia areata patients have higher levels of IL-6 and TNF-α.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

Both HLA-B and MICA are independently linked to alopecia areata.

Inhibiting TYK2 can restore hair growth in alopecia areata.

Deleting Snai2 and Snai3 causes fatal autoimmunity.

NLRP1 helps melanoma tumors grow by boosting inflammasome activation and reducing caspase-3 activity.