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Scientists found new and known long non-coding RNAs in mouse hair follicle stem cells that may be important for stem cell function and could be targets for cancer treatment.

The LMNA mutation affects skin structure even in asymptomatic carriers.

A new mutation in the human glucocorticoid receptor reduces its function and causes resistance to glucocorticoids.

Lrig1 marks a unique group of stem cells in mouse skin that can become different skin cell types.

Dermal fibroblasts have adjustable roles in wound healing, with specific cells promoting regeneration or scar formation.

ILK and ELMO2 help cells move and stick together, important for wound healing and hair growth.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Stem cell behavior varies with stimuli, and lineage changes can happen without affecting stem cell division.

The Lanceolate hair-J mutation in mice mimics human hair disorders like Netherton's syndrome.

A new mutation in the HR gene causes hair loss in a specific family.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

The research mapped diverse cell types in mouse lacrimal glands, aiding understanding of gland biology and diseases.

TNF-alpha inhibitors can cause various immune-related skin issues.

Lower GATA3 levels in mice help hair regrow by changing certain immune cells.

RANKL helps regenerate lung cells, potentially aiding lung repair in diseases like COPD.

MEX3A is crucial for maintaining intestinal stem cells in mice.

K6 gene expression can be controlled and manipulated in mice for studying skin disorders.

Folliculin deficiency causes problems with cell division and positioning due to disrupted RhoA signaling and interaction with p0071.

The STIM1 R304W mutation in mice leads to bone changes and teeth hair growth.

Different body areas have unique skin cell communication patterns, explaining why certain skin diseases occur in specific regions.

Certain immune system genes are linked to a higher risk of psoriasis and psoriatic arthritis, while others may offer protection.

TNC+ fibroblasts play a key role in skin inflammation by interacting with T cells.

A new mutation in the Hr gene causes hair loss in mice, similar to a human hair disorder.

A rare genetic mutation causes severe immune issues, hair loss, and nail problems.

Follistatin and LIN28B together improve the ability of inner ear cells in mice to regenerate into hearing cells.

Psoriasis patients' immune response to a hair protein depends on their specific gene type.

Lrig1 marks a unique group of stem cells in mouse skin that can become different skin cell types.