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New hair regrowth model proposed, imiquimod found to kill skin cancer cells, T-cadherin loss linked to invasive skin cancer, no clear gene link to skin cancer after transplant, and study on children's hair loss shows male dominance and genetic ties.

Human hair medulla doesn't break down hydrogen peroxide, which may affect hair color formation.

Certain immune cells are linked to non-scarring hair loss, suggesting potential for immune-targeted treatments.

Different skin stem cells help heal wounds, with hair follicle cells becoming more important over time.

Hair can naturally regain color after greying, and this change may be linked to stress levels.

Human melanocytes have unique traits that affect melanoma development and prognosis.

Wnt signaling is important for the change from the resting phase to the growth phase in human hair cycles.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

Nuclear shape and chromatin changes affect gene expression in skin cell differentiation.

Activating TLR3 may help produce retinoic acid, important for tissue regeneration.

Inactive hair follicle stem cells help prevent skin cancer.

Blocking cell death in hair follicles can lead to impaired hair growth.

Hair can naturally regain color, and stress might influence greying and its reversal.

The hair follicle structure is more complex than thought, with new findings on protein formation.

Hair follicle stem cells use specific chromatin changes to control their growth and differentiation.

Reactive oxygen species (ROS) influence hair growth by causing DNA damage, cell death, and changes in immune cells.

Mammoth hair from different ages shows distinct surface textures and elemental compositions.

Certain types of human hair might help ultraviolet rays reach and damage cells, possibly leading to melanoma.

A protein called MPZL3 in mitochondria slows down hair growth and could be a target for treating hair growth disorders.

Hair structural proteins are synthesized sequentially in specific cells, offering a new way to study hair proteins and defects.

A genetic mutation in the hHa1 gene creates a smaller, but still functional, hair protein without causing hair problems.

Different populations have distinct hair structures related to their ancestry.

DNA phenotyping can predict physical traits like eye, hair, and skin color, improving forensic investigations.

Miniaturized hairs stay connected to muscle in alopecia areata, allowing possible regrowth, but not in androgenetic alopecia.

A new mutation in the STING protein causes a range of symptoms and its severity may be affected by other genetic variations; treatment with a specific inhibitor showed improvement in one patient.

Hair follicles in androgenetic alopecia age faster, especially in the front.

Hair differentiation starts earlier than thought, involving multiple type-II keratins.

The gene p53 is crucial for removing damaged cells to allow for healthy tissue renewal.

Biomimetic peptides were found to promote hair growth and prolong the growth phase in human hair follicles.