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Monkeypox skin lesions show full-thickness skin death and swollen skin cells, with the virus found in affected cells.

ILC1-like cells may contribute to hair loss in alopecia areata.

Bullous pemphigoid is influenced by genetic factors, immune cell dysfunction, aging, and triggers, with treatment often improving symptoms.

Lichen planus commonly affects middle-aged adults, often involves limbs and mucosal areas, and can be linked to immune diseases.

Autoimmune polyglandular syndrome can cause temporary brain issues that may improve on their own.

Alopecia areata, a type of hair loss, is likely an autoimmune disease with a genetic link, but its exact cause is still unknown.

Three characteristics of plasmacytoid dendritic cells help tell apart lupus-related hair loss from LPP.

Contact immunotherapy can change immune responses in alopecia areata, suggesting new treatment targets.

Cytotoxic T cells cause hair loss in chronic alopecia areata.

In alopecia areata, certain immune cells increase and express a protein linked to immune activation.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

Consider infectious diseases like visceral leishmaniasis before diagnosing autoimmune hepatitis.

A man developed facial skin lesions after a stem cell transplant, which improved with specific treatments.

Skin and mucous symptoms are key for diagnosing autoimmune connective tissue diseases.

Netherton syndrome has two subtypes with shared immune traits but different allergic and immune responses, suggesting targeted treatments.

A woman had a rare skin condition with recurring painful nodules that heal in 6 weeks, often without needing treatment.

Immunosuppressive therapy helps manage autoimmune diseases but carries risks like infection and potential for malignancy.

Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

Early detection and histopathology are crucial to prevent permanent hair loss in cicatricial alopecia.

Purpura fulminans can signal underlying autoimmune disorders, not just infections.

Tyrosine kinases are important in skin autoimmune diseases and could be targets for new treatments.

IL-17C is important in inflammatory skin diseases and could be a target for treatment.

Relatives of lupus patients show signs of immune system activity but not full-blown lupus.

Antibodies help identify glycoproteins in normal skin and tumor cells.

Autoinflammatory skin diseases are complex, increasingly common, and significantly affect quality of life.

Fibroblast changes in systemic sclerosis may help understand disease severity and treatment.

DM and AA may share a common cause.