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A new skin disease in four Labrador retrievers responded well to immunosuppressive treatment.

PLAU and SerpinB2 affect cell death differently in various forms of leprosy and could be targets for new treatments.

Ancient immune and signaling pathways still regulate blood cell development.

Nail changes from immunotherapy can be managed without stopping cancer treatment.

Anti-MND antibodies are present in various diseases, not just PBC, and their levels don't correlate with disease activity or skin symptoms.

Alopecia areata is an autoimmune disease causing hair loss, treatable with immune-modulating drugs, and linked to genetics.

Cancer patients treated with immune checkpoint inhibitors may develop alopecia, but some hair regrowth is possible with treatment.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Immunotherapy for cancer caused a patient to develop a condition affecting hormone production, requiring ongoing hormone replacement therapy.

Four specific genes are linked to keloid formation and could be potential treatment targets.

IL-4 and IL-13 might play a role in a type of hair loss similar to alopecia areata.

HIV can cause skin disorders, which are often the first sign of infection, especially in people with darker skin.

IL-17C is important in inflammatory skin diseases and could be a target for treatment.

The antibody created from BCC tissues reacts similarly to both BCC and hair follicles, suggesting BCC may come from hair follicle cells.

A new mutation in the STING protein causes a disease with lupus-like symptoms and responds well to a specific inhibitor treatment.

Immunocompromised patients can develop skin and hair issues due to a virus.

Inflammation and immunity play a key role in androgenetic alopecia, with better treatment outcomes in certain immune-positive cases.

Animal models have helped understand hair loss from alopecia areata and find new treatments.

Alopecia areata involves complex immune responses, suggesting broader treatments could help.

Alopecia areata is an autoimmune condition causing hair loss, linked to genetic factors and immune system issues, with no cure yet.

Vδ1+ T-cells in the skin contribute to hair loss in alopecia areata and could be targeted for treatment.

An imbalance in gut bacteria is linked to skin immune diseases and may affect their outcomes and related health issues.

Alopecia areata involves complex immune activity, mainly Th1, with potential benefits from broader immune treatments.

Special and immunohistochemical stains are not routinely needed for diagnosing hair disorders.

ILC1-like cells can independently cause alopecia areata.

Hair growth phase in mice weakens certain immune responses.

Examining scalp tissue under a microscope helps diagnose and understand hair loss diseases.

Some drugs can cause skin reactions, which may improve when the drug is stopped, and rapid diagnosis and stopping the drug is crucial.

Children with alopecia areata have immune imbalances and are more likely to have certain health issues.

Biologic therapies can cause various adverse events, but allergy/immunology clinicians can manage them.