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ASLAN004 was safe and well-tolerated, supporting further development for treating certain diseases.

IL-18 signaling helps mature Tregs move into the thymus.

Chronic refractory alopecia areata has more skin-resident memory T cells, and JAK inhibitors may help reduce them.

T-cell patterns in skin help distinguish alopecia areata from androgenetic alopecia.

The new skin model can predict how chemicals might cause skin allergies.

Dual TCR Treg cells are common in various mouse tissues and show diverse characteristics.

Understanding how Regulatory T Cells work could help create treatments for certain skin diseases and cancers.

IL-17 plays a key role in severe hair loss in chronic alopecia areata.

The dense fine speckled pattern in ANA tests is common in autoimmune diseases, challenging previous beliefs that it was unrelated.

Children with frequent severe infections and low antibodies should be checked for immune deficiencies.

Biologic therapies can cause various adverse events, but allergy/immunology clinicians can manage them.

Tissue-resident memory T cells can protect against infections and cancer but may also contribute to autoimmune diseases.

Cutaneous lupus patients have higher levels of certain immune cells in their blood and skin.

Higher PD-1 levels are linked to fewer immune cells in hair follicles in alopecia areata.

Enhancing Tregs can protect against alopecia areata.

Patients with thyroid disorders show different symptoms and antibody levels.

Blocking IL-17 can reduce skin inflammation in a mouse model of pityriasis rubra pilaris.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

Psoriasis patients on biologic therapy have a weaker immune response to COVID-19 vaccination than healthy people.

Higher levels of IL-1R1 and hsa-miR-19b-3p may help diagnose and predict alopecia areata severity.

Leflunomide may reduce the risk of alopecia areata, while methotrexate, cyclosporine, and rituximab may increase it.

ILC1-like cells may contribute to hair loss in alopecia areata.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.

ILC1-like cells can cause alopecia areata by affecting hair follicles.

Patients with severe alopecia areata have higher levels of MIF, which decrease after successful treatment.

A mutation in the IKZF1 gene causes immune system overactivity, linked to autoimmune diseases like lupus.

Type-2 immunity may influence skin diseases and could be targeted for treatment.

NKG2D+CD4+ T cells are higher in alopecia areata patients and may be involved in the disease.

Combining hair transplantation with PRP is more effective for treating hair loss than hair transplantation alone.

People with Alopecia areata have higher levels of certain T regulatory cells in their blood.