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The length of the CAG repeat in the androgen receptor gene affects the risk and progression of prostate cancer, BPH, infertility, and undermasculinized genitalia.

A new genetic change causing early stop in the androgen receptor gene was found in a patient with androgen insensitivity syndrome.

Mutations in the androgen receptor gene cause androgen insensitivity, leading to female traits in genetically male individuals.

Testosterone increases certain kidney protein levels, potentially raising blood pressure.

Blocking androgen receptors early in life increases estrogen levels and reduces sexual motivation in male rats.

Different substances that activate or block the androgen receptor can affect male development and treat conditions like prostate cancer.

Sex steroids influence skin appendage function and neoplasm development, with changes in AR and ERalpha in certain gland neoplasms.

Androgen receptors play a key role in male development and prostate cancer, with treatments targeting androgen action.

Androgens may influence bladder cancer progression by affecting cellular behavior.

DHT deficiency may disrupt rat epididymis function by affecting estrogen receptors.

Red deer only have androgen receptors in neck hair cells for mane growth during breeding season.

Estrogen and androgen signals control synaptic changes in rat brains.

Two enzymes regulate androgen receptor activity, affecting treatments for androgen insufficiency and benign prostatic hyperplasia.

Androgens help motor neurons grow by increasing neuritin.

Early hormones shape sex-specific differences in rat glands.

Androgen receptor overexpression can increase prostate cancer cell growth even without hormones.

The same gene mutation can cause different symptoms in family members.

Sex steroid receptors are found in specific skin cells, affecting skin and hair functions.

Finasteride, a hair loss drug, may cause long-term sexual side effects due to changes in hormone receptor levels.

A woman's hyperandrogenism was caused by a genetic mutation leading to non-classic adrenogenital syndrome.

Certain changes to the B-ring of androgen receptor ligands can increase their effectiveness for potential treatments of muscle and bone conditions.

Hair sensitivity to androgens is partly controlled by specific enzyme expressions in different hair areas.

Women with non-classical congenital adrenal hyperplasia have higher levels of certain steroids, which can be reduced by treatment.

Androgens improve memory in adolescent male rats by acting on brain receptors.

Affected males are born with ambiguous genitalia, raised as females, but develop male traits at puberty due to enzyme deficiency.

Mutations in the androgen receptor gene cause Androgen Insensitivity Syndrome, affecting sexual development.

Androgens and estrogens are crucial for male reproductive health, affecting hormone levels and tissue function.

Two women with very high androgen levels had only slight skin issues, one due to a non-classical adrenal disorder and the other due to an adrenal tumor.

Mutations in the androgen receptor gene cause different levels of androgen insensitivity, making it hard to create simple tests for the condition.