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Testosterone replacement therapy, especially intramuscular, raises PSA levels, but no overall link between PSA and testosterone was found except in non-BPH patients.

Prostate cells have proteins that bind to specific hormones, which can increase protein production when activated by these hormones.

The normal human prostate does not significantly affect blood DHT levels.

Higher BMI and lower testosterone are linked to more aggressive prostate cancer.

The conclusion is that tritium-labeled testosterone metabolites can be made and are better converted into dihydrotestosterone in skin cells than in prostate tissue.

Testosterone increases acidification in the vas deferens by up-regulating V-ATPase.

Epitestosterone may act as a weak antiandrogen and can inhibit an enzyme involved in testosterone metabolism.

Testosterone helps human pancreatic cells increase insulin release.

Testosterone in our bodies helps increase insulin secretion from the pancreas.

Testosterone boosts fluid and electrolyte secretion in seminal vesicles, aiding sperm function.

Higher CRP levels suggest chronic inflammation is linked to bladder-related urinary symptoms in men with enlarged prostates.

Human prostate cells produce more WISP1/CCN4 when there's not enough oxygen.

Inhibiting testosterone 5alpha-reductase may help treat benign prostatic hyperplasia.

Testosterone can build muscle and bone without enlarging the prostate when a specific enzyme is blocked.

Testosterone makes the connections in the uterus lining simpler and lowers certain protein levels, which might lead to infertility.

Testosterone in human pancreas cells boosts insulin production.

Finasteride may improve prostate cancer therapy by enhancing testosterone's benefits and reducing risks.

The trial on testosterone replacement therapy in older men faced concerns due to prostate cancer risk.

Epitestosterone may counteract testosterone's effects and has roles in body processes like prostate growth and hair distribution.

Confirm testosterone levels with advanced tests to avoid unnecessary procedures.

The research found that androgens help control blood flow in the rat prostate through a specific binding site.

Dihydrotestosterone (DHT) promotes the growth and spread of aggressive brain tumor cells.

Incidental prostate cancer was found in 4.89% of patients undergoing surgery for benign prostatic hyperplasia.

Testosterone reduces key molecules needed for early pregnancy in rats.

Transgender men on testosterone therapy need ongoing gynecologic care due to potential cancer risks.

Blocking the enzyme that turns testosterone into DHT can safely and effectively treat enlarged prostate.