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Finasteride, at doses of 5mg or higher, may negatively affect male fertility by reducing the expression of certain genes involved in sperm production.

Disrupted stem cell signals in hairpoor mice cause hair loss.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

A mutation in the Adam10 gene causes freckle-like spots on Hairless mice.

HSD11b1 affects skin nerves and increases non-histaminergic itch.

Rac1 is crucial for normal hair structure and pigmentation.

Ovariectomized mice mimic postmenopausal hair loss, and estradiol helps maintain hair density.

SV40 T antigen in hair follicles causes abnormal hair and health issues in mice.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

Regulatory T cells are essential for normal and improved wound healing in mice.

A new mutation in the hairless gene causes hair loss and skin wrinkling in mice.

A mutation in the Sgkl gene causes defective hair growth in mice.

The type of tumor suppressor affects the form of skin cancer from hair follicle stem cells.

ISCK03 stops melanin production in human melanoma cells and lightens skin color in mice and guinea pigs.

Multiple pregnancies prevent skin disease but worsen kidney disease in certain mice.

The rhg mutation in mice affects the Oat gene, causing hair growth issues and other symptoms.

Akt2 and SGK3 are both important for normal hair growth and development.

Thrombospondin-1 is crucial for hair follicle regression and reduced blood vessel growth during the catagen phase.

The study found that a key immune pathway protecting hair follicles is reduced in a mouse model of scarring hair loss.

A specific DNA region is crucial for Foxn1 gene expression in thymus cells but not in hair follicles.

Cathepsin L deficiency causes hair and skin issues in mice.

Sox13 is a marker for early hair follicle development but not essential for skin and hair growth.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

The Trichodysplasia spinulosa virus protein can cause abnormal hair growth in mice.

Blocking JAK3 signaling can reverse hair loss from alopecia areata.

Reduced TRPS1 leads to increased STAT3 and SOX9 in hair follicles, affecting hair growth.

Hirosaki hairless rats have sparse, twisted hair due to missing hair keratin genes.

Female mice with disrupted 5α-reductase 1 had significant metabolic issues, including stress response problems, insulin resistance, liver fat buildup, and obesity.