Search

everythinglearnresearchcommunity

for

Search:↓

SETDB1 is essential for controlling DNA methylation, silencing retrotransposons, and maintaining skin cell health, with its absence leading to skin inflammation and hair loss.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

The N gene affects the protein makeup of mouse hair.

BMP-2 activates the Dlx3 gene in mouse skin cells, important for hair and skin development.

MAA beads improved wound healing in male mice by activating the Shh pathway, but not in females.

Interleukin 6 may help protect skin without causing inflammation.

A new method helps grow skin cells from humans and mice more easily and quickly.

Proteomic analysis can identify genetic differences in mouse hair, helping understand hair defects and variations.

Modulating BMP activity changes the number, size, shape, and type of ectodermal organs.

The assay effectively identifies compounds that affect immune cell activation.

The method allows for 3D tracking of hair follicle stem cells and shows they can regenerate hair for up to 180 days.

ODC transgenic mice can model human hair loss with skin lesions.

Optimized conditions and specific treatments enhance the growth of hair cells from C3H mice.

The Gsdma3 gene is essential for normal hair development in mice.

Changing the diet of mice lacking the enzyme CBS can affect symptoms related to the genetic condition.

Human and mouse TGase3 enzymes are similar but differ near the activation site, crucial for their function in skin and hair development.

Spiny mice have resilient, large mitochondria that help them regenerate tissue.

The p53 protein helps control hair follicle shrinking by promoting cell death in mice.

Genetically modified rats help reveal how vitamin D affects bone and skin health.

Sry may regulate fatty acid metabolism and shows different expression levels in rat tissues.

Tet2 and Tet3 enzymes are essential for controlling hair growth by affecting DNA demethylation and gene expression in mice.

The study maps how genes are regulated during mouse hair growth.

Skin tumor cells in patients with tuberous sclerosis have higher levels of a protein called cathepsin B.

Nonimmunogenic forms of keratins K71 and K31 can delay and prevent alopecia areata.

Noggin gene inactivation causes skeletal defects in mice, varying by genetic background.