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Matrical tumors share a common growth mechanism involving the Wnt pathway and consistent PHLDA1 expression.

aPKCλ is essential for hair follicle stem cell maintenance and wound healing.

Co5M offers a new way to observe and understand wound healing without labels.

Defects in certain proteins cause major heart abnormalities during early development.

Cupric chloride treatment corrected abnormal Purkinje cell development in brindled mice.

Keratins are crucial for cell stability, wound healing, and cancer diagnosis.

CDP is crucial for lung and hair follicle cell development.

High ODC and low K1 and K10 may indicate early skin tumors in mice.

Alk1 in specific cells is crucial for proper nerve branching and hair function.

A new mouse mutation causes skin and hair defects due to a gene change.

The microenvironment affects the behavior and survival of melanocytes with the GNAQ oncogene in melanoma.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

CaBP1 and CaBP2 are important for maintaining hearing by supporting continuous calcium currents and nerve signaling in the ear.

Elf5 is important for skin stem cell growth and could help treat skin and hair problems.

Keratin 15 cells from hair follicles help develop and maintain skin tumors in mice.

A specific gene segment can make mouse skin cells glow, helping study hair growth and gene effects.

Cathepsin E is crucial for normal skin cell differentiation and development.

The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

The tumor suppressor BRCA2 helps in cell division by bringing key proteins to the area where cells split.

The NF-κB effector p65/RelA activates hair keratin genes, aiding hair formation.

Protein Kinase Cε increases skin sensitivity to UV damage and skin cancer risk.

Scientists created a cell model to study and find treatments for a skin disease called RDEB.

A small change in the TRPS1 gene leads to a less severe form of a syndrome affecting hair, nose, and finger development.

AP-1 and TGFß work together to drive resistance in basal cell carcinoma, suggesting new treatment options.

The ventral matrix is the main source of the nail plate.

Keratin proteins are essential for keeping the cells in the human colon healthy and stable.