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Blue-light activation of TrkA improves hair-follicle stem cells' ability to become neurons and glial cells.

Testosterone significantly affects urination differences between male and female mice.

Key genes for mink fur have been identified, aiding conservation efforts.

The Kras mutation changes normal cell signals, leading to disrupted tissue structure and potential cancer.

The Naked gene in mice causes abnormal sebaceous glands and disrupts hair follicle organization.

The mouse model showed defects in adult stem cell maintenance related to Hutchinson-Gilford progeria syndrome.

KAP8.2 gene variations affect cashmere quality in goats.

KRTAP28-1 gene can help breed sheep with finer wool.

A new method can detect mutations in mice by observing changes in hair follicle cells.

Pigs without the Hairless gene showed skin and thymus changes, useful for studying human hair disorders.

Naked-mouse hair lacks certain proteins and has less soluble fibril.

The skin of both rat strains showed similar lectin binding patterns.

SK2 channels help control sensory signals in rat muscle spindles and hair follicles.

Overactive Stat3 in mouse skin causes hair loss and cell structure damage.

Dynlt3 is important for melanosome transport and skin coloration.

p120-catenin helps control skin inflammation by regulating cadherin levels.

A new type of pachyonychia congenita linked to a specific keratin gene mutation was found in two Pakistani families.

Protein Kinase Cε increases skin sensitivity to UV damage and skin cancer risk.

Trichocytic cytokeratins are found in hair, nails, tongue, and thymus cells, showing complex regulation in tissue development.

A mouse model was created to study hair loss similar to humans.

SkQ1 antioxidant improved health and lifespan in mice.

The tumor suppressor BRCA2 helps in cell division by bringing key proteins to the area where cells split.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

Lhx2 helps retinal cells respond to signals for eye development.

Vitamin D receptor knockout mice have significant motor impairments but no cognitive deficits.

Mouse models help study genetic skin diseases.

Blocking JAK-STAT5 signaling in mice leads to hair growth.

Patients with certain FoxN1 gene mutations have severe immune issues but normal skin and hair.