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Certain gene variations in Jiangnan cashmere goats are linked to important traits like birth weight and fiber quality, useful for breeding.

Overexpressing NSD3 in mice causes breast cancer-like tumors and gland abnormalities.

Nephronectin is linked to worse breast cancer outcomes and helps cancer spread.

A KRT32 gene variant causes loose anagen hair syndrome.

Basonuclin 1 (BNC1) helps skin cells multiply and move, which is crucial for wound healing.

Higher expression of the keratin-associated protein 8.1 gene in Liaoning cashmere goats is linked to finer cashmere fibers.

A specific DNA sequence caused hair loss in male mice by activating immune cells and increasing a certain immune signal.

Certain genetic variants reduce enzyme activity, contributing to non-classic congenital adrenal hyperplasia.

The microenvironment affects the behavior and survival of melanocytes with the GNAQ oncogene in melanoma.

A cluster of sulfur-rich hair protein genes was found on chromosome 17.

Keratinocytes can reverse the effects of the GNAQ oncogene, inhibiting melanoma cell growth.

Mutations in the HOXC13 gene cause hair and nail development issues.

Small molecules KY19382 and KY19334 may help treat skin cancer by reducing CDK1 levels and blocking harmful cell signals.

A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

FLCN helps control iron levels in cells.

The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

848 genes related to fat and metabolism are less active in people with Central Centrifugal Cicatricial Alopecia.

Clouston syndrome is inherited in an autosomal dominant pattern and caused by a specific gene mutation, with no current treatment available.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

A Jordanian family with Clouston syndrome has a common GJB6 gene mutation.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

A new genetic change causing early stop in the androgen receptor gene was found in a patient with androgen insensitivity syndrome.

A position effect on the TRPS1 gene causes excessive hair growth in humans and mice.

A rare genetic variant linked to skin cysts was found in blood DNA, suggesting its role in cyst formation.

A variant in the KRT31 gene causes a rare hereditary hair disorder called monilethrix.