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Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

Hoxc13 regulates specific hair protein genes on mouse chromosome 16.

K39 and K40 are the last keratins expressed in hair development, completing the hair keratin catalog.

Nanostructured lipid carriers effectively deliver tofacitinib to hair follicles, reversing hair loss in alopecia areata.

A new mutation in the KRT86 gene causes a hair disorder with variable expression.

Treating hair with 1M sodium hydroxide increases its friction, making it better for small medical knots.

A genetic variant in the KRT25 gene causes tightly curled hair.

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The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Two new proteins, hKAP1.6 and hKAP1.7, are found in the hair follicle cortex.

The lotion with N793 strain significantly increased hair density and reduced hair loss safely.

The Lanceolate hair-J mutation in mice mimics human hair disorders like Netherton's syndrome.

EGF and KGF affect wool fineness in Gansu alpine Merino sheep.

Polyquaternium-64 helps damaged hair look healthy again.

Krt16-deficient mice help understand skin disorders like PC and FNEPPK.

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Variant G of the KRTAP20-1 gene improves wool curliness in Chinese Tan sheep.

Farudodstat may effectively treat alopecia areata by protecting hair follicles without harmful effects.

Frontal fibrosing alopecia is a scarring hair loss condition mainly affecting postmenopausal women, with unclear causes.

Four-amino acid part makes enzyme sensitive to finasteride.

CDK4/6 inhibitors may cause hair loss in breast cancer patients.

Specific gene variants affect wool traits in Chinese Tan sheep.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.