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Circular RNA ERCC6 helps activate stem cells important for cashmere goat hair growth by interacting with specific molecules in an m6A modification-dependent way.

The curly mutation in SELH/Bc mice affects hair and may help study human genetic disorders.

K25, K27, and K28 are found in all inner root sheath layers of hair, while K26 is only in the cuticle.

Decreasing MIG6 can increase the movement and invasiveness of MEK-inhibited mutant NRAS melanoma, particularly when stimulated by EGF.

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Akt2 and SGK3 are both important for normal hair growth and development.

Mutations in the KRT16 gene can cause skin and nail disorders.

KRT2.13 gene is similar to KRT2.9 but not expressed in hair follicles.

Connexin-26 gene mutations may increase cancer risk in KID syndrome patients.

Akt2 protein is essential for normal cell division in early mouse embryos.

A gene variant in KRT71 causes the curly fur in Selkirk Rex cats.

CLK1 is needed for skin cells to become epidermal cells but not sebocytes.

A specific gene mutation causes different hair defects in Indian monilethrix families.

Keratin2-6g is crucial for hair follicle development, with mutations causing cell degeneration and vacuolation.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

Different conditions affect how hair proteins assemble, and certain mutations can change their structure.

Two new gene mutations cause a rare hair disorder.

Twist1 is crucial for UVB-induced skin cancer development.

Keratin 75 is important for fast wound healing and works with SOX2 and the LINC complex to help skin cells move and repair damage.

LGR5 and LGR6 are expressed differently in various skin tumors, which may offer clues about their origins.

The vector successfully directed specific gene expression in hair follicles.

Recombinant human TSG-6 speeds up wound healing in diabetic mice.

KRT72 gene helps form hair.

The B2 genes are crucial for hair growth in rats.

Integrin alphavbeta6 is important for wound healing and hair growth, and blocking it may improve these processes.

Over-expression of Sp2 can lead to cancer by preventing proper stem cell differentiation.

A substance called Compound 2g can strongly block STS (a hormone-related enzyme) without affecting estrogen levels, making it potentially good for treating breast cancer.