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Hair germ cells differ from epidermal cells in keratin expression, and specific keratins form after hair differentiation.

Using a combination of specific cell cycle regulators is better for safely keeping hair root cells alive indefinitely compared to cancer-related methods.

A Korean baby with nevus sebaceus syndrome was found to have a KRAS gene mutation.

High glycine–tyrosine keratin-associated proteins help make hair strong and maintain its shape.

Keratins K4 and K13 form stable dimers in mature esophageal cells, aiding cell stability.

Increasing m6A levels can improve skin cell growth and wound healing.

Triptolide effectively and safely reduces actinic keratosis lesions in mice.

A genetic variant in the KRT71 gene may cause loose anagen hair and wooly hair, and symptoms might improve with age.

Interleukin-1 increases keratin K6 production in skin cells.

Different keratins in hair follicles can help identify hair tumor origins.

Recombinant human TSG-6 speeds up wound healing in diabetic mice.

IGF2BP3 and other m6A-related genes are linked to keloid formation and could be potential treatment targets.

QLT0267 stops hair follicle cell growth and movement.

TRα and CRABPII genes change their activity levels during goat fetal skin development.

GRHL3 is important for controlling gene activity in skin cells during different stages of their development.

A gene variant causes a skin and hair disorder by disrupting protein balance.

Keratin patterns in hair follicles help understand hair growth and potential hair and nail disorders.

rhKGF2 significantly promotes hair growth and follicle development.

A new sheep gene, KRTAP36-1, may help breed sheep with better wool by reducing prickle factor.

A new mutation in the TRPS1 gene was found in a Ukrainian girl with Trichorhinophalangeal syndrome type I.

Kartogenin may help treat hair loss by promoting hair growth and extending the hair growth phase.

A new pair of mouse keratins, 65 kD and 48 kD, are found in specific skin areas and are linked to a unique skin differentiation type.

Different body areas have unique skin cell communication patterns, explaining why certain skin diseases occur in specific regions.

A new mutation in the TRPS1 gene caused Trichorhinophalangeal syndrome in a 17-year-old, highlighting the need for genetic testing.

Misdiagnosis led to unnecessary treatment due to a genetic mutation affecting thyroid hormone resistance.

Two specific SNPs in the TRPS1 gene cause excessive hair growth by altering the protein's structure.

Scientists created a cell model to study and find treatments for a skin disease called RDEB.

The KRTAP15-1 gene affects cashmere fiber thickness in goats.

A mutation in the KRT25 gene causes woolly hair and hair loss.