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A new pair of mouse keratins, 65 kD and 48 kD, are found in specific skin areas and are linked to a unique skin differentiation type.

Dermatologists should consider congenital syndromes like TRPS in young adults with early hair loss.

Sfrp2 increases during hair follicle catagen phase and slows keratinocyte growth.

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The mK6irs1/Krt2-6g gene likely causes wavy hair in mice.

mTORC2 is crucial for healthy skin barrier by regulating lipids and filaggrin.

Pachyonychia congenita is linked to a keratin gene on chromosome 17.

Keratin-based hydrogels with calcium are effective for delivering anti-fibrotic drugs.

Researchers created a cell line to study hair growth and found specific genes affected by dihydrotestosterone.

Different keratins in hair follicles can help identify hair tumor origins.

Shaykh Abbas Kuta Karang significantly advanced Malay medical knowledge with his detailed manuscript.

The vector successfully directed specific gene expression in hair follicles.

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CTIP2 may help in skin development and maintenance.

Keratins are crucial for cell structure, growth, and disease risk.

Matriptase is crucial for keeping epithelial tissues healthy and functioning properly.

Keratin 15 helps maintain skin cell growth and repair.

Keratin 17 gene mutations cause both steatocystoma multiplex and pachyonychia congenita type 2.

Activating PKM2 and Wnt/β-catenin signaling speeds up wound healing.

A child's scalp infection was caused by a fungus not previously known to do so, but it was successfully treated.

A new mutation in the TRPS1 gene caused Trichorhinophalangeal syndrome in a 17-year-old, highlighting the need for genetic testing.

MicroRNA-21 could help diagnose and treat skin fibrosis.

Bikunin is found on the cell boundaries of certain skin layers and may help regulate skin and hair functions.

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Genetic screening can help diagnose and manage infertility in Slovenian couples.

Skin cells might help spread prion diseases like Creutzfeldt-Jakob disease.

HtrA2 activity is crucial for normal hair growth by regulating fat cell development.