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Basal cell carcinomas in mice can start from hair follicle stem cells and other skin cell types, depending on signaling levels.

Certain immune cells may cause hair loss by reacting to stressed hair follicles.

Ossification in trichilemmal cysts is more common than previously believed.

Alpha 6 + /MHCI - cells have stem cell traits and are similar to mouse hair follicle stem cells.

Using micro skin tissue columns improves skin wound healing and reduces scarring.

Hair follicle cells help protect against immune attacks by regulating T-cell activity.

The FTO gene hinders stem cells in hair follicles from becoming pigment cells.

Tranilast successfully treated a man's skin sarcoidosis when other treatments failed.

Collagen XVII is crucial for preventing hair and pigmentation loss by maintaining melanocyte stem cells.

The study concludes that as skin matures from infancy to childhood, there are major changes in cell differentiation, stemness, and growth, leading to a stronger skin barrier in older children.

scMC effectively separates biological signals from technical noise in single-cell genomics data.

A child was initially wrongly diagnosed with a fungal scalp infection but actually had a non-scarring hair loss condition called Temporal Triangular Alopecia.

Mast cells may significantly contribute to central centrifugal cicatricial alopecia.

CD200 is not a reliable marker for identifying stem cells in all skin types.

Human dermal stem/progenitor cells can divide and differentiate more than hair follicle dermal papilla cells.

Researchers found a specific T cell receptor linked to severe drug reactions like Stevens-Johnson syndrome when patients take carbamazepine.

The local environment is crucial for cell development in the tongue.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Mesenchymal stem cells in people with Hidradenitis Suppurativa are more inflammatory, possibly contributing to the disease.

Langerhans cells and melanocytes migrate to the skin and hair follicles during early human development.

A new gene mutation may cause cyclic thrombocytopenia by affecting platelet production.

Metabolic issues may trigger lymphocytic cicatricial alopecia, and treatments targeting these issues could help.

Trichothiodystrophy hair is structurally abnormal with protein and organization issues.

Trametinib can effectively treat severe kaposiform lymphangiomatosis when other treatments fail.

DS cells in hair follicles can help form and restore hair, especially in hair loss conditions.

Recognizing LET and AITD can help diagnose SLE early for better treatment.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

CAR-T cell therapy shows promise for treating autoimmune disorders but faces challenges like complex manufacturing and limited tissue penetration.

Low TRPS1 expression in skin and hair cells is linked to hair problems in Trichorhinophalangeal syndrome.