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LPA3 signaling in the uterus is crucial for placental formation and fetal development.

PD‐L1 and PD‐L2 may not effectively control immune activation in alopecia areata.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Different enzymes are active in different parts of developing mouse organs.

Higher PHGDH levels cause unusual melanin buildup in hair follicles.

Both Th1 and Th2 immune responses are increased in alopecia areata, with Th2 response more strongly linked to how severe the disease is.

A humanized CXCL12 antibody may delay and treat alopecia areata by altering the immune response.

New genes and pathways are important for testosterone production and male sexual development.

Certain genetic variants reduce enzyme activity, contributing to non-classic congenital adrenal hyperplasia.

Your genes, especially IL-1a, can predict how well a hair growth treatment with platelet rich plasma will work for you.

ILC1 cells contribute to hair loss in alopecia areata.

The human keratin 6a gene's specific sequences trigger expression in skin layers after injury.

Boys with less severe EDA mutations in XLHED have milder symptoms and better sweat and hair production.

Gene therapy with a vitamin D receptor gene improved hair growth in rats with a type of rickets-related baldness.

Laminin-511 is involved in psoriasis development and can be regulated by cannabinoid receptor type 1.

CD4 T cells can cause alopecia areata by activating CD8 T cells to attack hair follicles.

GLABRA2 represses root hair formation by inhibiting a specific gene.

A49T gene variant linked to higher prostate cancer risk, lower hormone levels, and slightly reduced balding risk.

HB24 helps convert IBA to IAA, promoting root hair growth.

People with Alopecia areata have higher levels of prostaglandin D2 in their blood and tissues.

Restoring hair follicle immune privilege may help treat alopecia areata.

Low Vitamin D receptor levels in hair follicles may contribute to alopecia areata.

THA is a rare condition with no significant clinical consequences if thyroid function is normal.

No clear link between specific gene and hair loss in Mexican brothers.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.

SLHA can be hard to diagnose and needs teamwork between specialists.

The hepatitis B vaccine did not cause hair loss in the tested mice.

A mutation in the EDNRA gene causes Oro-Oto-Cardiac syndrome, affecting face and heart development.