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Mutations in the LSS gene cause hair loss and may affect brain development, with varying severity.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

New mouse models help study melanocytic cells for melanoma research.

A new mutation in the LIPH gene was found to cause a rare hair disorder in a Japanese boy.

LEF1 is essential for the development of airway glands and is regulated by the Wnt/ß-catenin pathway.

The LTF gene may help predict and manage nonspecific orbital inflammation.

The lentiviral array can monitor and predict gene activity during stem cell differentiation.

TLR2 helps control hair growth and regeneration, and its reduction with age or obesity can impair hair growth.

Krox20 is crucial for hair growth and maintaining skin stem cells.

The lanceolate hair-J mutation in mice helps understand human hair disorders like Netherton's syndrome.

Sox13 is a new marker for early hair follicle development and differentiation.

LGR5 and LGR6 are expressed differently in various skin tumors, which may offer clues about their origins.

TLR2 is important for hair growth and can be targeted to treat hair loss.

Researchers found that the Leptin receptor is a consistent marker for hair follicle dermal cells, which may help future hair research.

Overexpression of ΔNLef1 in mouse skin leads to hair loss, cysts, and skin tumors.

Hoxc13 gene expression and skin thickness change similarly during cashmere goat hair follicle development.

Hox proteins help maintain keratinocyte identity by regulating miRNA expression.

A specific type II hair keratin was identified and found in hair cortex and tongue cells.

Lnc056 helps hair follicle stem cells grow by increasing TRIP6 expression.

FoxN1 overexpression in young mice harms immune cell and skin development.

The BMP/Smads pathway and Id2 gene control hair follicle stem cells, affecting their rest and growth phases.

QLT0267 stops hair follicle cell growth and movement.

The mouse hairy ears mutation causes longer ear hair due to changes in gene expression.

EGM2 and SOX2 help form beige adipocytes by maintaining ASC immaturity and activating brown adipocytes.

Suppressing the HGPS mutation may improve symptoms and suggest reversibility.

Blocking CXCR4 may help treat hidradenitis suppurativa.

New LSS gene variants help understand congenital hypotrichosis 14 better.

The assay effectively identifies compounds that affect immune cell activation.

GRHL3 is important for controlling gene activity in skin cells during different stages of their development.

RNase L hinders hair follicle regeneration by altering immune signals.