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The SLICK1 allele in Holstein heifers affects hair and immune traits without altering prolactin signaling.

The LMNA mutation affects skin structure even in asymptomatic carriers.

Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

Blocking JAK1 or JAK3 helps reverse hair loss in a mouse model of alopecia areata.

Lichen planopilaris causes permanent hair loss and scarring due to damage to hair follicles and can be mistaken for other hair loss conditions.

Deleting a specific protein in skin cells disrupts normal hair growth and development.

Mouse touch-sensitive nerve cells adjust their connections based on competition with other similar cells.

Resolving malaria-related lung issues doesn't rely on Alox12 and leaves some inflammation.

Total glucosides of paeony may help treat alopecia areata by reducing inflammation and hair loss.

The gene causing hair loss and heart issues in rough coat mice is still unknown.

Blocking JAK3 signaling can reverse hair loss from alopecia areata.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Sphingosine 1-phosphate helps control mechanical pain.

RXRα is crucial for proper immune response and links diet to immune function.

Removing alkaline phosphatase from human skin cells hinders the creation of new hair follicles.

The PON1 192R gene variant is linked to a higher risk of psoriasis and heart disease in Western Mexico.

Platelet-rich plasma may successfully treat lichen planopillaris, as shown by one patient's symptom regression.

GLABRA2 represses root hair formation by inhibiting a specific gene.

A gene mutation worsens skin irritation in mice due to a lack of certain fats.

Toll-like receptor 3 helps repair the skin barrier after UV damage.

Mice with human-like EGFR had growth issues, skin defects, heart problems, and unusual bone development.

PPAR-γ agonists like pioglitazone may help manage lichen planopilaris but don't fully reverse scarring.

Mrp3 may aid in wound healing and hair growth.

TTNPB helps turn stem cells into neural stem cells, improving depression-like behaviors in rats.

Lipin-1 is important for skin cell differentiation and skin barrier function.

A position effect on the TRPS1 gene causes excessive hair growth in humans and mice.

Specific immune cells and pathways contribute to hair follicle inflammation and hair loss, suggesting potential treatments for lichen planopilaris.

Specific mutations in a receptor cause facial abnormalities and hair loss.

Different types of PPARγ are found in varying amounts in human skin and its parts, which could affect how skin treatments work.

Mutations in β1 integrins cause embryonic death but have milder effects on skin.