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Hair color production in mice is closely linked to the hair growth phase and may also influence hair growth itself.

Lanceolate complexes in mouse hair follicles are essential for touch and depend on specific cells for maintenance and regeneration.

Understanding gene expression in hair follicles can reveal insights into hair growth and disorders.

Fibronectin and other basement membrane components increase during hair growth and decrease during rest.

Retinoic acid causes gland formation instead of hair in mouse skin by altering epidermal and dermal interactions.

Actin filaments help stabilize and integrate cell membranes during transfer.

The mouse hairy ears mutation causes longer ear hair due to changes in gene expression.

Lhx2 is a crucial regulator of the Sonic Hedgehog signaling in early mouse retinal development.

Lhx2 is essential for effective Sonic Hedgehog signaling in early retinal development.

Birds can regenerate inner ear cells using specific gene pathways, unlike mammals.

Targeting adipocyte-to-mesenchymal transition could help treat fibrosis.

EGM2 and SOX2 help form beige adipocytes by maintaining ASC immaturity and activating brown adipocytes.

Mice can regrow hair on wounds due to specific cell interactions and mechanical forces not seen in rats.

Restoring cell communication can treat tissue disorders.

Restoring cell communication can treat tissue disorders.

Iron-dependent hormones help wheat roots adapt to temperature stress.

MIM is crucial for hair follicle formation and regeneration by controlling cilia formation and hedgehog signaling through its interaction with Cortactin and Src.

Murine skin wounds become less stiff over time as they heal.

Gene regulatory regions evolve faster than protein coding regions, allowing new gene relationships without changing transcription factors.

TGF-β signaling is essential for new hair growth after wounds.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

Pig skin cells can turn into mesodermal cells but lose their ability to become neural cells.

Inhibiting Bmp signaling disrupts hair growth and differentiation.

High levels of ERK activity are key for tissue regeneration in spiny mice, and activating ERK can potentially redirect scar-forming healing towards regenerative healing in mammals.

Changes in keratin make hair follicles stiffer.

Nerve cells and other cell types work together to start horn growth in dairy goats.

The method quickly analyzes hair growth genes and shows that blocking Smo in skin cells stops hair growth.

Both main and alternative Wnt signaling are important for regrowing rodent whisker follicles.

Sweat glands and hair follicles are determined by opposing signals, with BMPs promoting sweat glands and blocking BMPs leading to hair follicles.