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Overexpressing Merkel cell virus proteins in human hair follicles can create clusters of cells that resemble Merkel cell cancer.

Merkel cell polyomavirus can infect and persist in skin cells, evading the immune system, but certain treatments can control it.

Beta-HPV and MCPyV are linked to certain skin cancers, with ongoing research and vaccine development.

The study concluded that the developed models are effective for studying hair growth mechanisms and testing new treatments.

Longer immunotherapy treatment may improve outcomes for Merkel cell carcinoma patients.

A young woman with a rare skin cancer was diagnosed late because her symptoms were unusual for the disease.

Merkel cell carcinoma is most likely to recur within two years of diagnosis, and factors like immune suppression, being over 75, and male sex increase this risk.

Notch signaling stops Merkel cell development, and hair follicles help maintain them after skin injury.

Merkel cells develop independently of nerves and are linked to specific hair follicles in mice.

Atoh1 expression can create new Merkel cells in the skin.

Sensory neuron remodeling and Merkel-cell changes happen independently during skin maintenance.

Sensory neuron changes and Merkel-cell changes in the skin happen independently during normal skin maintenance.

Sensory neuron and Merkel cell changes in the skin happen independently during normal skin maintenance.

Sensory neuron remodeling and Merkel-cell changes in the skin happen independently.

Sensory neuron and Merkel-cell changes in the skin happen independently during normal skin maintenance.

Sensory neuron and Merkel cell changes in the skin happen independently during normal skin maintenance.

FGFR2 signaling controls Merkel cell formation in different skin regions.

Surgery and antifungal medication are effective for treating Merkel cell carcinoma with fungal infection.

BMP signaling is essential for the development of touch domes.

Merkel cells stabilize nerve endings in the skin, and they change independently of each other.

Sensory neurons and Merkel cells remodel at different rates during normal skin maintenance.

Sonic hedgehog signaling is needed for the development of touch-receptor cells in the skin, and the loss of Polycomb repressive complex 2 can lead to more of these cells.

Merkel cells are abundant in facial vellus hair follicles, especially during the anagen phase.

Merkel cell increase is specific to certain skin diseases, not general skin growth.

BerEP4 and CD34 staining can help tell apart tricholemmoma from basal cell carcinoma.

Some skin growths with mucin can form hair follicles and resemble skin cancer, but a special stain can help tell them apart.